Author
1. Shah PK
ISBN
9789350252758
DOI
10.5005/jp/books/11124_106
Edition
1/e
Publishing Year
2013
Pages
9
Author Affiliations
1.
LTM Medical College and LTMG Hospital Sion, Mumbai-400 022, India,
Seth GS Medical College and KEM, Hospital, Mumbai, Maharashtra, India,
Seth GS Medical College, KEM Hospital, Mumbai, Maharashtra, India,
Seth GS Medical College and KEM Hospital, Mumbai, Maharashtra, India; FOGSI 2012,
KEM Hospital, Mumbai, Maharashtra, India,
Mumbai,
King Edward Memorial Hospital and Seth Gordhandas Sunderdas Medical College, Mumbai, Maharashtra, India,
Lokmanya Tilak Municipal General Hospital, Mumbai,
Seth GS Medical College and King Edward VII Memorial Hospital, Mumbai, Maharashtra, India; Fogsi 2012,
Mumbai, Maharashtra, India,
Seth GS Medical College and King Edward VII, Memorial Hospital, Mumbai, Maharashtra, India,
Cedars Sinai Medical Center, Los Angeles, California, USA,
KEM Hospital, Parel, Mumbai, Maharashtra, India,
Seth GS Medical College, Mumbai, Maharashtra, India,
KEM Hospital, Mumbai, Maharashtra, India; FOGSI 2012,
Seth GS Medical College and KEM Hospital, Mumbai, Maharashtra, India,
LTMG Hospital, Mumbai,
Chapter keywords
Vaso-occlusive disease,
atherosclerosis,
lipoprotein infiltration,
leukocyte adhesion,
proinflammatory priming,
proinflammatory gene program,
proinflammatory signaling,
ligand-receptor interactions,
vascular cell adhesion molecule 1,
cytokines,
smooth muscle cell proliferation,
oxidation,
atherosclerotic lesions,
plaque disruption,
macrophage colony stimulating factor,
fibroproliferative lesion,
atherogenesis,
vasoconstrictor,
Toll-like receptors,
subendothelial retention,
chemotactic cytokines,
plaque angiogenesis,
necrotic lipid core,
ST-elevation myocardial infarction,
matrix-degrading metalloproteinases,
gelatinases,
cell-to-cell interaction,
spotty microcalcification,
atheromatous lesion,
endothelial erosion,
interleukin-6,
serum amyloid A,
necrotic core,
fatal coronary thrombi,
C-reactive protein,
abrupt luminal occlusion,
inflammatory cascade,
symptomatic vascular disease,
cell infiltration,
tenascin-C,
cathepsins,
apoptosis
