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Chapter-184 What the Adult Cardiologist Should Know about Cyanotic Congenital Heart Disease?

BOOK TITLE: CSI: Cardiology Update 2014

Author
1. Rao P Syamasundar
ISBN
9789351526186
DOI
10.5005/jp/books/12415_185
Edition
1/e
Publishing Year
2015
Pages
8
Author Affiliations
1. UT-Houston Medical School, Houston, Texas, USA, P.Syamasundar.Rao@uth.tmc.edu, University of Texas-Houston Medical School, Houston, Texas, USA, University of Texas, Houston Medical School, Children’s Memorial Hermann Hospital, Houston, Texas, USA, University of Texas-Houston Medical School Children’s Memorial, Hermann Hospital, Houston, Texas, USA, University of Texas-Houston, Medical School, Houston, Texas, USA, University of Texas-Houston Medical School, Children’s Memorial Hermann Hospital, Houston, Texas, USA, University of Texas–Houston Medical School, Houston, Texas, USA, University of Texas at Houston McGovern Medical School, Houston, Texas, USA, University of Texas-Houston McGovern Medical School/Children’s Memorial Hermann Hospital, Houston, Texas, USA
Chapter keywords
CHD, Cyanotic heart defects, tetralogy of Fallot, transposition arteries, tricuspid atresia, truncus arteriosus, pulmonary venous connection

Abstract

CHD is a malformation of the heart and developed during intrauterine period. CHD is generally classified as acyanotic and cyanotic. Cyanotic heart defects are tetralogy of Fallot, transposition of the great arteries, tricuspid atresia, truncus arteriosus and total anomalous pulmonary venous connection. Tetralogy of Fallot is a collection of four abnormalities to include ventricular septal defect, PS, right ventricular hypertrophy and dextroposition of the aorta includes residual shunts, arrhythmias etc. Transposition of the great arteries includes atrial switch operation and rastelli operation. Truncus arteriosus a common arterial trunk arises from base of the heart and the truncus gives rise to the aorta, pulmonary artery and coronary arteries. In TAPVC all pulmonary veins are connected to common pulmonary vein which in turn drains into the left innominate vein, superior vena cava, coronary sinus and portal vein. Tricuspid atresia is characterized by congenital absence of the morphologic tricuspid valve. Palliated cyanotic CHD related to late detection because of negligible symptomatology secondary to balanced circulation.

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