Textbook of Medical Mycology Jagdish Chander
INDEX
Note: Page numbers followed by f refer to Figure and t refer to Table.
A
Abdominal mucormycosis
signs of 562
symptoms of 562
Abscesses 621
Absidia corymbifera See Lichtheimia 556, 571
Acacia nilotica 298
Acanthamoeba 858
AccuProbe 90
Acervulus 52, 912
Achorion schoenleinii 162
Acid production 418
Acid-fast stain, modified 212f, 860
Acquired immunodeficiency syndrome 740
Acremonium 640, 683, 769, 781, 791, 912
falciforme 204, 217, 694
kiliense 694, 781
species 640, 648, 687, 781
Acridine orange stain 866
Acrodontium 791
Actidione 449, 912
Actinobacillus
capsulatus 709
lignieresi 218
Actinomadura 863
madurae 204206, 209f, 210f, 216
pelletieri 204, 205, 216, 217
Actinomyces 860, 863
Actinomycetoma 204t, 206, 206t, 209f, 211f, 214f, 215f, 216t, 218
grains of 212f
of foot 210f
of shoulder 209f
Adamson's fringe 166
Adenosine triphosphate 885
Adhesive tape technique 869
Adiaconidia 35, 708, 709t, 912
Adiaspiromycosis 707, 708, 711, 714
Adiaspirosis 707
Adiaspore 35, 912
Aerial hyphae 27
Aerobic actinomycetes 216
Aeromycology 14
Aerosolized pentamidine 482
Aflatoxicosis 763, 766
Aflatoxin 765, 912
African histoplasmosis 322
Agaricus bisporus 770
Ajellomyces 313
capsulatus 48, 313
dermatitidis 41, 50f, 343, 709
Aleurioconidia 709, 912
Aleuriospore 912
Algae-like organisms 54
Allergen immunotherapy 728
Allergic
alveolitis, extrinsic 722, 725
aspergillosis 526, 737
Aspergillus sinusitis 737
bronchopulmonary
aspergillosis 526, 723, 725, 737
mycoses 725, 697
diseases 405, 409
forms 527
fungal
diseases 722
rhinosinusitis 726
sinusitis 737
manifestations 409
respiratory diseases 722t
rhinosinusitis 146
Allescheria boydii 683
Allochthonous 912
Allylamines 103, 107, 108
Alphacel-yeast extract agar 853
Alternaria 640, 723, 740, 785, 789
alternata 279, 726, 789
dianthicola 279
infection 275
species 32, 640, 724f, 785, 785f, 787
tenuissima 279
Amanita 773
muscaria 771
pantherina 771
phalloides 771
proxima 772
pseudoporphyria 772
smithiana 772
virosa 771
American Type Culture Collection 891
Ameroconidium 912
Amido black solution 85f, 871
Amine alkaloid 766
Aminoadipic acid 798
Amphotericin B 104, 242, 674
colloidal dispersion 105
conventional 104
lipid complex 105
lipid-based formulations of 105
liposomal 105
Amsterdam declaration 42
Anamorph 912
Anamorphic fungi 52
Aneuploid 912
Angular cheilitis 405, 408
Animal
disease 230
inoculation techniques 872
pathogenicity 86, 137, 151, 188, 218, 241, 262, 284, 304, 320, 349, 367, 388, 422, 481, 516, 538, 575, 647, 650, 673, 713, 719, 747, 805, 820, 837
Annellide 912
Annellidic Scopulariopsis species 913
Annelloconidia of Magnusiomyces capitatus 620f
Annelloconidium 913
Annular erythematous scaly plaques 170f
Antheridium 913
Antibacterial therapy 63
Antibiotics 106
Antibody
detection of 84, 421, 537
Antifungal 104f, 108, 111, 114, 877
agents 165, 481
antibiotics 103
classification of 103
disks 882
drugs 284, 368, 454, 877
amphotericin B 881f
classification of 103t
resistance 115
practice guidelines 115
susceptibility testing 874
therapeutic drug monitoring 116
therapy 102, 117
Antigen 441
culture filtrate 832, 837
detection of 84, 367, 537
heat-labile 366
Anti-neutrophilic response 60
Antiretroviral therapy 717
highly active 314, 497
Antiretroviral treatment 517
Antler hypha 913
Antrodia camphorata 769
Apophysis 913
Apophysomyces 555, 558, 561, 563, 567, 568, 781, 836, 853, 913
causes 561
elegans 40, 46
genus 557
infection 561
mexicanus 555
morphology of 568
ossiformis 555
species 556, 557, 567, 568, 572
trapeziformis 555
variabilis 50f, 555, 569f
Apothecia 913
Appressoria 913
Aquatic fungi 829
lower 30
Arterial catheters 63
Arthritis 405, 408
Artifacts 75, 135, 180, 447, 858, 872, 873t
Arthroconidia 28f, 360, 913
Arthroderma 163
vanbreuseghemii 44
Arthrographis 696
kalrae 696
Arthrospores 157f, 185f
chains of 188f
Ascomycetes 33, 50f, 204
Ascomycetous yeasts 30
Ascomycota 47, 50, 146, 313, 343, 715
phylum 53, 230
Ascospore 50f, 913
Ascostromata 913
Ascus 913
Aseptate 913
Asexual
conidia 604
propagule 436
reproduction 31
sporulation 567
Aspergilloma 526, 527, 527f
Aspergillosis 85f, 524, 527t, 528, 684
types of 531t
Aspergillum 524
Aspergillus 5, 26, 27, 50, 63, 71, 72, 81, 84, 110, 189, 524, 528, 538, 614, 640, 670, 687, 723, 725, 727, 737, 739, 762764, 768, 848, 851, 880, 910, 913, 915, 916, 918, 920
alliaceus 671t
antigens 537
candidus 671
clavatus 525
endocarditis 529
flavus 525, 531, 533, 534f, 671, 725, 738, 740, 766, 782, 913
fumigatus 46, 65, 422, 524, 525, 528, 531, 533, 538, 539, 622, 647, 671, 724726, 737, 740, 748, 782, 920
antigen 527
culture of 527
septate hyphae of 535f
fungal ball 533, 539
genus 538, 640
glaucus 525, 536
infections 524, 525, 528
meningoencephalitis 528
nidulans 13, 204, 525, 531, 536, 622, 671, 916
microscopic appearance of 537f
niger 525, 531, 536, 538, 671, 674f, 768, 782
hyphae of 672f, 674f
mycelial growth of 673f
ochraceus 768
oryzae 525, 766
sections 52
sinusitis 737
species 5, 26f, 80, 114, 209, 513, 525, 526, 529, 530, 531f, 533, 534f, 538, 538t, 539, 622, 648650, 651f, 671, 675, 781
septate hyphae of 76f, 531f533f
tamarii 766
terreus 110, 525, 528, 531, 539, 671
colony morphology of 536f
tracheobronchitis 527
versicolor 525, 528, 671, 766, 916
Asteroid body 229, 232, 913
Asthma
bronchial 526, 527, 723
severe 723
Asymptomatic oral carriage 408
Athlete's foot, tinea pedis 174
Atovaquone 482
Atrophic erythema, acute 408
Auditory canal, external 671f, 672f
Aureobasidium 640
melanogenum 149
pullulans 279, 640, 785, 785f
species 785
Autophagy 65
Autospore 913
Auxacolor testing 417f
Auxanographic method 853
Auxarthron 791
Auxenochlorella protothecoides 806
Azelaic acid 130
Azole 103, 108
action of 108
antifungal 349
B
Bacilliform 913
Bacteria 31t
Bacterial agents 204
Bagassosis 727
Baker's yeast 12, 621
Balamuthia 858
Balanitis 406, 408
Balanoposthitis 406
Balkan endemic nephropathy 768
Ballistoconidium 913
Bamboo rats, species of 512
Bandicota bengalensis 510
Barber's itch 172
Basal ascomycetes 53
Basal media 847
Basidiobolomycosis 554, 556, 597, 598t, 603
Basidiobolus 597, 834
ranarum 556, 558, 562, 575, 603, 605, 835, 917
culture of 606f
sexual spores of 606f
Basidiomycetes 33, 53, 155, 231
Basidiomycetous
fungus Schizophyllum commune 51
yeasts 30
Basidiomycota 47, 50, 128, 155, 231
phylum 53
Basidiospore 51f, 913
Basidium 913
Beauveria species 683
Beauvericin 769
Beigel's disease 154
Benzylamines 103, 107, 108
Bifonazole 109
Bioassay technique 117
Biochemical tests 418
Biological warfare 10
Bioluminescence assay 885
Biopsy 499
frozen-section 79
Bioterrorism 360
Biotin-avidin amplifications system 537
Bipolaris 271, 640, 723, 739, 740, 785, 787, 788, 915
hawaiiensis 726, 785
species 280f, 640, 785
spicifera 614, 740, 785
Bird seed agar 850
Bitunicate pyrenomycetes 252
Biverticillium 507
Black piedra 158
Black yeast 27, 146, 147f, 257, 279, 281, 785
Blackish pasty colonies 147f
Blakely's red gum 439
Blankophor 858
Blastic conidial development 34
Blastoconidium 913
Blastomyces 343, 613, 709, 713, 715, 716, 791, 870
dermatitidis 24, 25, 36, 80, 81, 114, 302, 342347, 347f, 348, 348f, 349, 350, 366, 380382, 440, 648, 650, 709, 713, 719, 791, 803, 804, 848, 849, 851, 853, 871
antigens 349
culture 348f
strains 343
yeast 344
gilchristii 342, 343
loboi 297
percursus 342, 343, 716
species 695
Blastomycetic dermatitis 342
Blastomycin 349
Blastomycosis 342, 346, 347
disseminated 345
queloidiforme 297
types of 346
Blastoschizomyces capitatus 619
Blastospore 453, 913
Blood
agar 851
plate 836f
cells 405
culture 74
glucose-cysteine 318
stream infections 402
Blurry vision 498
Body fluids 74
Bone marrow 74
Bonsai trees 231
Botryomycosis 218
causative bacteria of 218t
Brachiola 495
Brain
abscess 146
heart infusion agar 719, 849
Brazilian blastomycosis 379
Brewer's yeast 12, 621
Bronchoalveolar lavage 531
Broth dilution method 876
Broth macrodilution 877
method 877
Brownish mycelial colonies 187f
Brudzinski's sign 443
Bull neck 384
Burow's solution 112
Busse-Buschke disease 434
Butenafine 107
Byssinosis 727
Byssochlamys 768
C
‘C’ or ‘S’ -shaped streaks 643
Caenorhabditis elegans 87, 872
Caffeic acid agar 850
Calcium binding protein 314
Calcofluor white stain 858
Canavanine glycine bromothymol blue (CGB) agar 854
Cancer 65
Candida 5, 24, 63, 72, 81, 82, 113, 114, 401, 440, 538, 640, 641, 722, 805, 871
albicans 7, 27, 34, 40, 65, 80, 83f, 114, 241, 401403, 405, 406, 411, 413f, 415, 416t, 418, 640, 641, 648, 849, 854, 868, 914
antigens 71, 403
nonspecific 421
auris 402, 416, 669
fungemia 416
dubliniensis 27, 402, 415, 416t, 849, 850, 854, 868
enolase antigen testing 421
fukuyamaensis 12
genus 422
glabrata 316, 317, 402, 403, 414, 415, 418, 854
guilliermondii 402, 415, 418
haemulonii 402, 403, 416
complex 416
infection 41, 63, 177, 406, 407, 420, 672
kefyr 402, 415
krusei 402, 403, 408, 413, 418, 640, 854
growth of 414f
lusitaniae 402, 410, 416
pancreatitis 409
parapsilosis 46, 156, 402, 403, 414, 418, 803, 854
growth of 414f
pelliculosa 416
rugosa 402, 416
score 423
species 5, 26f, 27, 36, 65, 78f, 83, 87, 105, 110, 111, 178, 188, 404, 410, 418, 422, 423, 513, 530, 615, 649, 650, 670, 675
growth of 413f
mixed infection of 412f
pseudohyphae of 410f413f
virulence factors of 404
stellatoidea 411
strains 420
tropicalis 402, 403, 413, 418, 640, 854
growth of 414f
viswanathii 402
Candidal granuloma 405, 407
Candidemia 405, 408
Candidial meningitis 408
Candidiasis 401, 405t, 407t, 409
Candiduria 407
Cannomys badius 507, 509, 510
Capilliconidia 913
Capnodiales 146
Capsular polysaccharide 441
Carbohydrate
assimilation media 853
fermentation media 853
Carbon
assimilation technique 136
dioxide 240
fermentation technique 136
Cardiogenic pulmonary edema 479
Castaneda's method 849
Castellani's method 889
Castellani's paint 112
Cave disease 315
Cell
mass vaccine 838
mediated immunity 85, 382
membrane 30
of fungi 30f
structure 29
titan 920
Cell wall 29
antigens 403
components 421
composition 31
mannoprotein 421
Cellobiose 452
Cellular immune responses 382
Cellular immunity 61
Cellulose 813
Centers for Disease Control and Prevention 891
Central bronchiectasis 527, 724
Central nervous system aspergillosis 528
Cephalosporium 694
acremonium 12
Cerebral mycosis 274
Cerebral phaeohyphomycosis 272
Cerebrospinal fluid 74
Cerinosterus cyanescens 231
Cetacean lacaziosis-like disease 302
Chaetomella raphigera 37
Chaetomium 786, 918
atrobrunneum 275, 280
globosum 280
perlucidum 280
species 252, 274, 280, 785, 786f
Champagne cup appearance 569f
Champagne glass 568
Champignon mushroom 770
Chemoprophylaxis 423
Chicago's disease 342
Chignon disease 154
Chitin synthase 29
Chitosomes 29
Chlamydia pneumoniae infections 316
Chlamydospore 914
Chlamydospore formation 417
Chlorella 797, 806, 914
Chlorellaceae 798
Chlorellales 797, 798, 806
Chlorellosis 798
Chlorophyll, lack 798
Chlorophyta 798
Chloroplast 914
Chorioretinitis 649
CHROMagar Candida 83f, 414f, 418
medium 854
CHROMagar Malassezia 854
Chromatography, thin layer 772
Chromoblastomycosis 146, 251, 252, 252t, 255f, 258f
Chromomycosis 251
Chrysosporium 163, 695, 695f, 696, 740, 781, 791
articulatum 695
parvum var.
crescens 695, 708
parvum 695, 708
species 347, 386, 683, 791, 791f
zonatum 695, 696f
Chytridiales 811
Chytridiomycetes 811
Chytridiomycota, phylum 55
Ciclopiroxolamine 111
Circinate 914
Clades 917
Cladistics 914
Cladogram 914
Cladophialophora 280
bantiana 16, 271, 272, 280, 280f
carrionii 252, 253, 258, 261f
Cladosporium 32, 257, 258, 723, 740, 912, 919
bantianum 280
carrionii 258, 280
castellanii 146
cladosporioides 156, 912
species 284, 726, 787
trichoides 280
type 258
of sporulation 259f
Claviceps 763, 766
fusiformis 766, 767
purpurea 13, 766, 919
Cleistothecia 50, 51f, 537f, 914
Cleistothecial ascomata 53
Clinical Laboratory Standard Institute (CLSI) 874
Clotrimazole 109
cream 675
Coagulase-negative staphylococci 218
Coccidia 474
Coccidioidal meningitis, chronic 362
Coccidioides 319, 356, 357, 360, 363, 366, 613, 791, 801, 812, 818, 870, 915, 919
immitis 14, 17, 46, 356, 357, 364, 365, 707, 791, 792, 871
posadasii 14, 114, 356, 365, 791, 792
species 11, 27, 28f, 36, 80, 209, 350, 357, 360, 364f, 365f, 366368, 510, 648, 713, 915
infections 114
spherules 365f
Coccidioidin 366
Coccidioidoma 361
Coccidioidomycosis 356, 362, 910
disseminated 361
Coccidium seeberia 811
Coelomycetes 52, 53, 269, 918
pycnidia of 29f
Cokeromyces 919
recurvatus 386, 556, 558, 568
Colletotrichum 646, 688
coccodes 646
dematium 640, 646f
gloeosporioides 646
species 646
truncatum 646
Columba livia 439
Columella 914
Combating fungal infections 895
Commercial mushroom 770
Complement fixation test 367
Complement receptors 404
Conidia 27, 32, 34, 535f, 709, 914
chains of 690f, 692, 692f, 693f, 783f
multiple 718f
type of 270
Conidial ontogeny 34
Conidiobolomycosis 554, 556, 597, 598t, 599, 600f, 601f
swelling of 601f
Conidiobolus 834
coronatus 556, 599, 601, 602, 835
broad hyphae of 603f
culture of 602f
papillated conidia of 603f
villose conidia of 603f, 604, 604f
incongruous 556
sexual spores of 604f
lamprauges 556
sexual spores of 604f
species 606
Conidiogenesis 34
types of 34
Conidiogenous cells 788f, 789
Conidiophores 32, 535f, 914
Conidium 914
Coniosporium epidermidis 149
Contagious disease 358
Continuous ambulatory peritoneal dialysis 801
Copper-penny 253
Coprine atrementarius 770
Coprine poisoning 770
Coprinus syndrome 770
Coprophilous fungi 6
Coremia 783, 914
Coremium 914
Cornmeal agar 848
Cornmeal-tween agar 849
Cornea 645f
Corneal abscess 642f
Corticosteroids, adjunctive 482
Cortinarius 772
orellanoides 772
orellanus 772
rubellus 772
speciosissimus 772
Corymbs 569
Corynebacterium 156
diphtheriae 409
minutissimum 179
tenuis 157, 179
Coturnixcoturnix japonica 271
Counterimmunoelectrophoresis 388
Coxiella burnetii 316
Creatinine-dextrose-bromothymol-blue-thymine 452
Cryptic fungal species 52
Cryptic species 229, 914
Cryptococcaceae 128, 155
Cryptococcemia 444, 453
Cryptococcal meningitis 443
Cryptococcoma 443
Cryptococcosis 434, 446
Cryptococcus 5, 24, 51, 63, 72, 81, 110, 111, 113, 188, 436, 440442, 451, 805, 848, 850, 854, 859, 913, 914
adeliensis 438
albidus 435, 437, 438
gattii 435, 854
complex 438
immune response 441
laurentii 435, 438, 641
liquefaciens 438
neoformans 14, 16, 59, 65, 80, 363, 434, 435, 437t, 438, 439, 453t, 648, 818, 850, 851, 880
budding yeast cells of 449f
species 438
species 27, 29, 76, 77f, 78, 78f, 79f, 347, 386, 437, 439, 444, 451, 456, 621, 649, 803
budding yeast cells of 450f, 451f
virulence factors of 441
yeast-like mucoid growth of 451f
strain 451
Cryptosporidium hominis 861
Cryptostroma corticale 727
Cryptostromosis 726
Culicinomyces clavosporus 13
Cunninghamella 780, 919
bertholletiae 556, 569, 570f, 575, 781
species 558, 780
Cunninghamellaceae 556
Curvularia 271, 640, 739, 740, 912, 915, 918
geniculata 217, 787
infections 281
lunata 281f, 647f, 648, 726
pallescens 270
species 280, 640, 646, 787
Cutaneous aspergillosis 529
Cutaneous blastomycosis 345
Cutaneous cryptococcosis 443, 444
primary 445
Cutaneous emergomycosis 716
Cutaneous histoplasmosis 315
Cutaneous lesions 155
Cutaneous mucormycosis 559, 561
extensive primary 562f
Cutaneous mycoses 11
superficial 125
Cutaneous phaeohyphomycosis 272
Cutaneous protothecosis 800
Cutaneous pythiosis 833
Cutaneous rhinosporidiosis 816
Cutaneous ulcerated plaque, primary 362f
Cyanobacteria 43
Cyclopeptides 771
Cyclopiazonic acid 766
Cyclospora cayetanensis 861
Cygnus 814
atratus 814
olor 814
Cylindrical conidia 283f
Cylindrocarpon 688
cyanescens 782
destructans 782
lichenicola 782
lucidum 13
species 782, 782f
Cyst 475
Cysteine-heart 849
Cystic subcutaneous
phaeohyphomycosis 274f
Cystoisospora belli 861
Cytoplasmic antigen 404
enzyme immunoassay 420
Cytoplasmic fusion 32
Cytoplasmic membrane 30
Cytosine deaminase 111
Cytosol 30
Cytotoxic chemotherapy 345
Czapek-Dox agar 851
D
Dactylaria 269
Dalmau plate culture 848
Dandruff 133
Dapsone 482
Dasypus novemcinctus 382, 389
Dauer cell 798, 914
Dematiaceous fungi 146, 146t, 251, 270, 270t, 739, 780, 784
Dendrogram 914
Denticles 240
Deoxyribonucleic acid
bar coding 90
probes 367
Dermatitidis 342, 345
Dermatitis 133
Dermatomycoses 146, 162
Dermatophilosis 171
Dermatophyte 28f, 163, 166, 169t, 176, 182f, 183, 880
arthrospores of 181f
causative 166t
conidia of 181t
ecological classification of 164t
septate hyphae of 181f
species 32f
spiral hyphae of 184f
test medium 160, 182, 851
Dermatophytic mycetoma 210
Dermatophytosis 162
chronic 166
deep 177
Dermoscopy 148
Descemet's membrane 640, 641
Detoxification 774
Deuteromycetes 128, 146, 204, 230, 252, 269, 358, 709, 914
Deuteromycota 34, 51
phylum 53
Diabetes mellitus 7
Diacetoxyscirpenol 768
Diaminodiphenylsulphone 820
Diaminopimelic acid 798
Diaper dermatitis 407
Dichloran rose-bengal chloramphenicol 850
medium 850
Dichotomous 531, 564
Diff-Quik stain 862
Digital object identifier 908
Dimethyl sulfoxide 856, 877
Dimorphic fungi 709
Diploid 915
Diplorhinotrichum gallopavum 269
Dipodascus capitatus 619
Dirofilaria 712
Disjunctor cell 915
Disk diffusion method 881
Dixon agar, modified 852
Dolipores 915
Dothideomycetes 146
Double ring sign 275
Doughnut-lifting sign 601
Drechslera 740, 787, 788
Drosophila melanogaster 87, 872
Dulcitol 452
Dusty cotton disease 727
Dystrophic onychomycosis, total 177
E
Echinocandins 113
Econazole 109, 675
Ectothrix 915
infections 168
Eczema, atopic 133
Eiken latex agglutination test 452
Emerging fungal
diseases 9
infections 622
Emergomyces 707, 709, 714719
africanus 707, 714716
orientalis 346, 707, 714
pasteurianus 44, 316, 513, 707, 714, 715, 718f
growth of 718f
species 42, 343
Emergomycosis 4, 36, 42, 44t, 309, 316, 322t, 346, 347t, 513, 637, 707, 714, 915
Emmons’ modification 848
Emmonsia 707710, 714, 715
crescens 343, 707711, 713, 715, 716
in vitro growth of 713f
parva 710
var. crescens 695, 708
var. parva 695, 708
parvum 708
pasteuriana 707, 714, 715
species 80, 364
Emmonsiella 313
capsulata 313
Emmonsiellopsis coralliformis 709
Encephalitozoon
cuniculi 495, 496, 500
hellem 496
intestinalis 494498
Endemic mycoses 9, 358
Endemic nephropathy 768
Endocarditis 405, 408
Endocystic cells 356
Endogenous endophthalmitis 648
Endomyces species 12
Endophthalmitis 405, 408, 639
fungal agents of 648t
Endophyte 915
Endoreticulatus species 496
Endoscopic sinus surgery 745, 749
Endospores 813, 819, 915
Endosporulation 915
Endothrix 915
infection 168, 168f
Enniatins 769
Enolase 421
Enterocytozoon bieneusi 494, 495, 496, 497
Entomogenous fungi 6
Entomophthorales species 554, 556, 604t, 890
Entomophthoramycosis 556, 597, 599f, 835t
Environmental protection agency 501
Enzymes 404
Eosinophil count, absolute 747
Eosinophilia 527, 724
Eosinophilic fungal rhinosinusitis 739
Epicoccum
nigrum 787
species 785, 787, 787f, 789
Epidemiological cut off 881883
Epidermophyton 889
floccosum 28, 112, 173, 174, 183, 187f, 671
species 163, 185
Epithelioid histiocytes, masses of 233
Epithelioid macrophages 712f
Epithet 915
Epizootic lymphangitis 322
Epsilometer test (Etest) 883
Equine phycomycosis 834
Ergometrine 766
Ergosterol quantitation method 885
Ergot alkaloids 766
Ergotamine 767
Erythematous annular
borders 175f
lesions 172
Erythrasma 180f
Escherichia coli 218
Esophagitis 408
Etymology 915
Eucalyptus camaldulensis 439
Eukaryota 29, 475
Eukaryotes 46, 47
Eukaryotic organisms 29
Eumycetoma 146, 203, 206, 206t, 214f, 215t, 218
black grain 204
grains 211
of foot 211f
Eumycota 24
Euphractus sexcintus 304
European blastomycosis 434
European Committee on Antimicrobial Susceptibility Testing 874
European Society of Clinical Microbiology and Infectious Diseases 874
Exoantigen 837
tests 871
Exogenous endophthalmitis 648
Exophiala 257, 850, 880
dermatitidis 40, 150, 252, 262, 279, 281, 281f
genus 284
jeanselmei 204, 215, 216, 279, 281, 282f
species 257, 284, 913
spinifera 252, 279
werneckii 283
Expiratory volume, forced 724
Exserohilum 271, 740, 787
rostratum 271, 726
conidia of 282f
species 282, 640, 788
Extensive hypopigmented patches 132f
Extensive nodulo-plaque lesions 255f
Extrapulmonary pneumocystosis 478
Eye 75
Eyeball 639
Eyeglass-shaped cells 239
F
Facial swelling 600f
Fantastic fungi 773
Farm fungicide 115
Farmer's lung
causative agents of 723t
disease 722
Favic chandelier 180, 915
Faviform 915
Favus 166, 167, 915
Fetal bovine kidney, primary 772
Feulgen reaction 862
Filamentous bacteria 216
Gram-positive 212f
Filamentous fungi 647, 878
Filamentous septate hyphae, long 746f
Filobasidiella
bacillispora 436
neoformans 436, 442
of basidiomycetes 51f
Fine needle aspiration cytology 713, 818
Fission 25, 915
yeast 915
Five-kingdom classification
animalia 47
fungi 47
monera 47
plantae 47
protista 47
Five-kingdom system 46
Floccose 915
Florida horse leeches 834
Flowcytometry 884
Fluconazole 109, 189
Flucytosine 111, 675
Fluorescein dye 643f
Fluorescence
activated cell sorting 884
antibody, direct 480
in situ hybridization 813
resonance energy transfer 89
Fluorescent
antibody stain 866
antibody staining 80
brightener wet mount 76f
septate hyphae 531f
Foamy histiocytes, sheets of 318f
Focal pulmonary lesions 453
Folliculitis 134
Fonsecaea 257, 258, 640
compacta 252, 258, 261f
pedrosoi 251, 252, 253, 254, 258, 259, 259f, 640, 918
species 284
Food
adulteration, prevention of 773
and agriculture organization 773
Foot cell 781, 525, 915
Formazan 815
Fourier transform infrared 803
Freeze-drying 890
Fumonisins 767
Fungal
agents 204, 215
causative 204
antigens 86t
cell 422
target sites of 104f
wall polysaccharides 30t
corneal ulcer 6, 639, 641, 642f645f
perforated 642f
culture 80, 135, 149, 180, 239, 257, 279, 303, 318, 347, 365, 386, 411, 449, 481, 500, 514, 567, 643, 650, 673, 713, 717, 745
collections 891
drying of 890
interpretation of 82
media 847
debris 749f
dimorphism 35
disease 7t, 59, 86, 302
burden of 8
classification of 11
diagnosis of 71
magnitude 6
re-emerging 9
endophthalmitis 647
genera 257
granuloma 529f, 530f
immunity 65
immunogens 60t
infection 5, 17, 60t, 63t, 93t
encountered 103
healthcare-associated 622
index of suspicion 17
of cornea 639
outbreaks 17
study forum 904
surveillance of 14
isolates, identification of 82
keratitis 639
mass 671f
metabolites, detection of 538
morphology 24
pathogens, common 60t
reagents 856
registries 19
repositories 18
rhinosinusitis 737, 741t, 742f, 745f, 747f, 748f
categorization of 738
classification of 738t
slide culture 82, 869
stains 856, 876
taxonomy 8, 39
vaccines 64
Fungi 3, 10, 30, 31t, 43, 128, 494, 770, 915, 811, 829, 830t
Absidia-like 569
allergenic 11
ball 527, 527f
cell wall of 30f
classification of 46, 52t
culture collections of 888
dimorphic 27, 35, 36, 60, 299
ecology of 6
egg 829
epidemiological markers of 92
harmful properties of 13
imperfecti 1, 34, 48, 52, 914, 917
taxonomy of 34
opportunistic 60
plasmalemma of 30f
preservation of 888
rectangular arthrospores of 32f
reference laboratory 891
septate hyphae of 26f
significance of 12
test 883
testing laboratory 892
true 203
useful properties of 12
yeast-like 176
zoological 44
Fungicide 915
FungiScope 19
Fusaproliferin 769
Fusariosis 687
Fusarium 26, 189, 614, 640, 683, 739, 740, 762, 763, 764, 769, 848, 880, 915, 919
chlamydosporum 689f
culmorum 769
graminearum 769
oxysporum 178, 687, 688
sacchari 646f
solani 640, 687
species complex 687
species 79, 110, 204, 218, 533, 640, 642, 645, 648, 683, 689f
sporotrichioides 769
verticillioides 687, 767
Fuseaux 916
Fusiform 916
G
Galactose 452
Gall bladder 499
Galleria mellonella 87, 872
Ganoderma lucidum 769
Gas
liquid chromatography 772
production 418
Gastric mucormycosis 562
Gastrointestinal basidiobolomycosis 605
Gastrointestinal mucormycosis 559, 561, 562
Genome sequence typing 622
Genus
Apophysomyces 555
Aspergillus, ascospores of 51f
Basidiobolus 606
Cryptococcus 436, 437
Cunninghamella 557
Glomerella 646
Malassezia, taxonomy of 128
Mucor 556
Rhizomucor 556
Saksenaea 557
Geomyces 696
pannorum 696
Geotrichosis 618
Geotrichum 615, 790, 913
candidum 618f, 696
hyphae of 619f
yeast of 618f
capitatum 619
species 157, 615, 619, 619t, 789
Germ tube 413f, 916
test (GTT) 417, 868
Giant cells 414 916
of granuloma 712f
Giant cutaneous 816
Gibberella 769
Giemsa staining 277f, 616
Gilchrist's disease 342
Gilled mushroom 770
Glabrous skin 170f
Glenosporella loboi 297
Glenosporopsis amazonica 297
Gliocladium roseum 13
Global Action Fund for Fungal Infections (GAFFI) 8
Glomeromycota 47, 48, 554, 555
phylum 53
Glucose 418
yeast extract 357
Glycoprotein 477
major surface 477
Gomara 131
Gomori's methenamine silver stain 863
Graft-versus-host disease 613
Grain eumycetoma, white 204
Gram's smear 410
Gram's stain 860
Granular bacteriosis 218
Granular dermatitis 834
Granule 916
Granuloma
trichophyticum 169
tricofitico 162
Granulomatous
disease, chronic 622
mycoses 64
reaction 214f, 254, 511, 748f
type of 233
Graphium 683
eumorphum 31
species 686f
Greenish mycelial colonies 690f
Greyish white mycelial colonies 187f
Gridley's fungal stain 863
Griseofulvin 106, 675
microsize 189
Grocott-Gomori's methenamine 78
silver 742
Guiana dolphins 300
Guillermo seeber 811
Guinea pigs 188
Guizotia abyssinica 82, 850, 851
agar 438
Gymnothecia 916
Gymnothecium 916
H
Haemophilus influenzae 59
Hair 162
bait technique 869
invasion, types of 169t
perforation test 182, 868
Hairbrush sampling technique 182
Hairy pus 561
Haloprogin 113
Hamycin 106
Hansenula anomala 614
Hansenula polymorpha 13
Haploid 916
Haplomycosis 707, 708
Haplosporangium 708
bisporale 707
parvum 707, 708
Helianthus annuus 451
Helicobacter pylori infection 12
Helicostylum lucknowense 573
Helminthosporium species 787, 788
Hematogenous dissemination 408
Hematoxylin stain 861
Hemoglobin agar 849
Hendersonula toruloidea 149, 187
Herbarium 916
Heterokaryon 916
Heterotrophs 3
Hilum 916
Histo spots 316
Histoplasma 312, 318, 360, 613, 695, 709, 713, 715, 791, 870
capsulatum 14, 16, 36, 47, 48, 60, 65, 75, 80, 81, 241, 302, 311314, 317f, 320, 321f, 343, 346, 347, 366, 381, 383, 513, 648, 649, 713, 714, 717, 727, 792, 849, 851, 853, 871
mycelial colonies of 312f
var. duboisii 853
species 318, 319, 386, 920
Histoplasmin skin test 319
Histoplasmosis 311, 319f
capsulati 314
chronic 319
classic 322
disseminated 316
duboisii 321
farciminosi 322
Holomorph 916
Homothallic fungi 27
Honeycomb-like adiaconidia 712f
Hormodendrum pedroso 251
Hormographiella 696
aspergillata 683, 696, 762
species 683
Horta's contribution 145
Hortaea
acidophila 146
werneckii 145148, 150, 150f, 151, 272
Hospital infection 622
Hulle cells 537, 916
Human
infection, natural 507
rectal tumor 812
Humoral immunity 61
Hyaline 916
fungi 269
hyphomycetes 640, 741, 780, 781
septate hyphae 718f
Hyalohyphomycetes 687t
Hyalohyphomycosis 682, 783
type of 524
Hydrolytic enzymes, secretion of 404
Hydrophilic fungus 300
Hydrophilic protistan 811
Hymenomycetes 890
Hypercalcemia 347
Hyperkeratosis of
fingers, diffuse 174f
palm, diffuse 174f
Hyperpigmented patches 132f
Hypersensitivity
delayed-type 319
pneumonitis 722
reactions 527t
Hyphae 157f, 417f, 620f, 916
broad, non-septate 602f
Hyphochytrium 55
Hyphomyces destruens 829
Hyphomycetes 52, 53, 146, 230, 252, 269, 358, 709, 890
Hyphomycosis destruens 828, 829
Hypopigmented scaly lesions 131f
Hypopyon 916
I
Index Fungorum 45
Ibotenic acid 771
Iceberg, tip of 716
‘Id’ reactions 722
candidiasis 409
dermatophytosis 169, 173, 178
Idiopathic orbital inflammatory disease 816
Ignis sacer 767
Imidazoles 108
Immune reconstitution inflammatory syndrome 62, 314, 455, 717
Immunity 59, 130, 165, 205, 232, 253, 272, 300, 344, 360, 440, 477, 497, 510, 525, 558, 710, 799, 814, 832
acquired 59
mechanisms of 59
Immunization 483
Immunofluorescence assays 480, 500
Immunohistochemistry 85
Immunology 83
Immunotherapy 423, 838
Imperfect fungi 916
In vitro tests 86
India ink stain 857
Indian childhood cirrhosis 766
Indian Council of Medical Research 892
Indian Medlars Centre 910
Infection
healthcare-associated 613
single source 622
Infectious diseases 405
Inocybe 771
fastigiata 771
Inositol 452
Insoluble antifungal drugs, list of 877t
International Agency for Research on Cancer 765
International Society for Human and Animal Mycology 738, 798, 904
Interstitial plasma cell pneumonia 473, 476
Intestinal microsporidiosis 498
Intestinal mucosa 499
Intracellular Histoplasma capsulatum 318f
Intracranial Aspergillus
granuloma 529f
Intracranial mucormycosis 564f
Intracystic bodies 475
Intranasal steroids 728
Invasive aspergillosis 527, 738
Invasive disease 738
Invasive fungal infections 613
Irregular septate
branching hyphae 279f
hyphae 277f
Isavuconazole 111
Itraconazole 110, 189
J
Jock itch 173
Juvenile type of disease 383
K
Kaposi's sarcoma 479
Karyogamy 32, 916
Kelley's agar 318
Keloidal blastomycosis 297
Keratinomyces 163
Keratinous debris 671
Keratitis 916
Keratomycosis 146, 639, 640t, 647t, 916
nigricans palmaris 145
Kerion 166, 167, 916
celsi 167
Kernig's sign 443
Ketoconazole 109, 675
Khaki-colored mycelial growth 187f
Kidneys 499
Killing yeasts, in vitro 344
Kingdom Chromista 55
Kingdom fungi 53t
classification of 47t
Kinyoun's method 860
Kinyoun's staining 211
Klebsiella pneumoniae 59, 441, 857
Knot body 28
Kodsi's classification 406
Kodua poisoning 766
Kunkers 832, 834
L
Lacazia loboi 44, 201, 299, 300, 303, 382, 828
budding yeast of 303f
Lacaziosis 44, 297, 302
like disease 297, 302
Lactophenol cotton blue
mount 869
stain 858
Lactose 418, 452
Lagenidiosis 828, 838
Lagenidium 797, 828, 838, 839
ajelloi 838
albertoi 838
giganteum 834, 838, 839
species 828
Lamellar keratectomy 647
Lanose 916
Laryngeal disease 346
Laryngitis 408
Latex particle agglutination 367
Leading International Fungal Education (LIFE) 8
Lecythophora hoffmannii 670
Legionella 316
Leishmania donovani 311, 316, 511, 514, 717
Leptosphaeria senegalensis 204
Leukoplakia 408, 409
Lichen planus 409
Lichtheimia 558, 569, 781, 913
corymbifera 44, 556, 558, 561, 562, 569, 570f, 571, 575
ramosa 569
species 568, 569
sporangia of 557f
Life-threatening disease 304
Liposomal-encapsulated amphotericin B 105
Liquid chromatography
high performance 117
high pressure 772
Liquid gold, tears of 131
Liver 499
Loboa loboi, See Lacazia loboi
Lobo's disease 297, 298
Lobomyces loboi 297
Lobomycosis 297
Loculoascomycetes fungi 252
Lomentospora 614, 683, 685, 687
prolificans 44, 684, 687f
Lowenstein-Jensen medium 216, 851
Lung cavities 361
Lutz's mycosis 379
Lutz-Splendore-de Almeida disease 379
Lycoperdonosis 727
Lymphadenopathy 347
Lymphatic lesions 384
Lymphocutaneous sporotrichosis 233, 233f, 234f
Lymphoma 477
Lyophilization 890, 916
M
Macroconidia 32, 32f, 183f186f, 312
clubbed-shaped 187f
formation, types of 182f
sickle shaped 689f
spindle-shaped 186f
Macroconidium 916
Macular scaly lesions 131f
Madura foot 203
Maduramycosis 203
Madurella 204, 851
grisea 215, 216
mycetomatis 204, 212, 215
culture of 216f
mycelial growth of 214f
species 64
Magnusiomyces 615
capitatus 44, 615, 619, 619t, 620f
hyphae of 619f
pseudohyphae of 619f
species 619
yeast-like colonies of 620f
infections 614, 619
species 615
Majocchi's dermatophytic granuloma 170f
Majocchi's granuloma 162, 169, 210
Malassezia 42, 51, 128, 138, 155, 670, 852, 859, 918
allergens 134
arunalokei 128
dermatis 128, 129
folliculitis 134f
furfur 125, 127129, 137, 179, 650
globosa 128, 129, 136, 854
japonica 128, 129
nana 129
pachydermatis 129, 134, 889
slooffiae 128, 129
species 36, 129t, 135, 136f, 137, 138, 614
colonies of 136f
sympodialis 128, 129
yamatoensis 128, 129
Malasseziaceae 128
Malasseziales 128
Malasseziomycetes 128
Malasseziosis 127, 130
Malbranchea 366, 781, 791, 792, 915
species 791, 792f
Malt extract agar 852
Maltose 418, 452
Manihot esculenta 814
Maple bark Stripper's disease 726
Mariner's wheel 299
Mass spectrometry 117
Masson-Fontana
silver staining 864
stain 278
Maxillary sinus 737
right 749f
Mayer's mucicarmine 819f
stain 864
May-Grunwald Giemsa 819, 861
stain 861
Medicopsis romeroi 44, 205
Medlar bodies 251, 253, 919
Medical Mycology, AIDS 10
in India 17
Meiosporic fungi 48
Melanin 441, 865
production 441
role of 64
Melbourne Code 45
Melibiose 452
Meningitis 405, 408
Meristospore 606, 916
Merogony 496, 498
Merosporangia 573, 781
Merosporangium 557, 916
Mesocricetus auratus 304, 388, 506
Mesomycetozoea 54, 811, 813
Metabolites, detection of 421
MIC testing 879
Miconazole 108, 151
Microbial Type Culture Collection 892
Microconidia 32, 32f, 183f186f, 270, 312
formation, types of 182f
Microconidium 916
Microcycle conidiation 917
Microdilution 877
Microfungus collection 891
Microspore, phylum 54, 494
Microsporidia 474
Microsporidial keratoconjunctivitis 498
Microsporidiosis 494, 499
Microsporidium 8, 54, 495, 858, 860
ceylonensis 495, 496
Microsporum 28, 912
audouinii 28, 168, 169, 179, 183
canis 28, 65, 165, 168, 169, 179, 183, 186f, 188, 650, 671, 868
distortum 165, 179
ferrugineum 165, 179
gypseum 32f, 163, 167, 169, 179, 186f, 188, 783
infections 168
persicolor 164
species 178, 183, 210
Miliary 347
coccidioidomycosis 362
Miltefosine 114
Mimicking blastomycosis 347t
Mimicking diseases 347
Mimicking tinea nigra, chemical dye 148f
Mites, control of 793
Mitospore 917
Mitosporic fungi 34, 48, 52
Molecular
beacons 90
methods 241
taxonomy 40
techniques 537
tools 188
typing 538
Molluscum contagiosum-like lesions 512f
Moniliaceae 709
Moniliasis 401
Moniliform 917
Moniliformin 769
Monod's sign 527
Monokaryon 917
Mononucleate 708, 713
Monosporium apiospermum 683
Morpholines 112
Mortierella 53t, 829
wolfii 567
Morula 798, 802
Mucocutaneous
candidiasis, chronic 406
histoplasmosis 315
lesions 384
Mucoid 620f
Mucomembranous Aspergillus bronchitis 723
Mucor 27, 556
circinelloides 386, 556, 564
racemosus 556, 917
species 49f, 563, 571, 781
sporangia of 557f
Mucorales 554, 556
Mucormycetes 26f, 30, 33, 72, 412f
fungi 780
non-septate hyphae of 76f, 78f, 565f, 566f
Mucormycosis 85f, 554556, 575, 835t
agents 80
Mulberry-like erosions 384
Multi-drug resistant infection 607
Multilocus enzyme electrophoresis 137
Muriform 256f, 257f, 785
bodies 251, 254, 917
cells 252, 253, 269
conidia 724f, 785f
macroconidia 270
Muscarine poisoning 771
Muscle 499
Mushroom poisoning 769
Mycelia 348f, 917
sterilia 35, 53
Mycelial colonies
black 259f
white 184f
Mycelial fungal strain 870
Mycelial fungi 876
Mycelial growth 718f
Mycelial-to-yeast conversion, in vitro 348
Mycelium 26, 917
Mycetism 762, 769
Mycetismus 762, 769, 770
Mycetoma 41, 203, 214, 917
classification of 206
grains of 213f
of foot 207f, 208f
of hand 208f
Mycetomata 917
Mycobacterium
leprae 303
tuberculosis 851
ulcerans 605
Mycobiome 917
Mycobiota 917
MycoBank 45
Mycocladus corymbiferus See Lichtheimia 556, 571
Mycological cure 77
Mycological techniques, conventional 868
Mycology 128, 158, 163, 230, 252, 495, 682, 708, 714, 740, 770, 917
Mycopathologia 380
Mycophagy 770, 917
Mycoplasma pneumoniae 316
Mycorrhiza 917
Mycosis 9, 146, 613, 637, 722, 762, 917
deep 11
fungoides 3, 356
healthcare-associated 622
mucorina 554
of ocular adnexa 650
superficial 11, 72, 146
Mycotic
diseases 36, 36t
granuloma, organized mixed 254
keratitis 639
Mycotoxic porcine nephropathy 768
Mycotoxicoses 762, 764
Mycotoxin 762, 770
conference 763
detection of 772
prevention and control of 763
Myelogenous, chronic 477
Myospherulosis 712, 917
Myrothecium 769
N
Naegleria 858
Naftifine 107
Nail 162
plates, destruction of 176f
Nannizziopsis vriesii 696
Nasal
cavity 749f
crust, debris of 29f
lesions 347
rhinosporidiosis 815
National AIDS Control Organisation 509
National Collection of Pathogenic Fungi (NCPF) 891
yeasts cultures 891
National Committee for Clinical and Laboratory Standards 874, 876
National Culture Collection of Pathogenic Fungi (NCCPF) 892
National Fungal Culture Collection of India 892
National Informatics Centre 910
Nattrassia mangiferae 270, 282, 920
Necrotizing reaction 511
Neoscytalidium
dimidiatum 44, 178, 187, 187f, 188f, 282
hyalinum 188
species 188
Neuro-paracoccidioidomycosis 385
Neurospora crassa 13
Neutral red stain 859
Neutrophil
chemotaxis 242
count, absolute 738
Niger seed agar 850
Nigrosin stain 857
Nigrospora
conidia of 788f
species 788
Nikkomycin 114
Nivalenol 768
Nocardia 209, 851, 860, 863
asteroides 204, 205, 216, 217, 851
brasiliensis 204, 205, 216, 217
caviae 204, 205, 216, 217
otitidiscaviarum 217
species 206, 209f, 211, 212f
Nocardiopsis dassonvillei 204, 216
Nodular organ 28, 180, 917
Nodules consisting of hyphae 157f
Nodulo-ulcerative lesions 233, 233f, 234f
Non-albicans Candida species 402, 416, 422
Non-cardiogenic pulmonary edema 479
Non-dermatophyte fungi 176
Non-dermatophytic
dermatomycosis 178
fungal species 187
Non-immune host factors 63
Non-invasive
colonization 527
disease 738
Non-neoformans Cryptococcus species 437, 454
Non-septate hyphae 554, 557f, 565f, 602, 835f
Non-sporulating mold 283
North American blastomycosis 342
Nosema
bombycis 497
ocularum 496
Nosocomial
candidiasis 409
fungemia 622
infections 622
Nova plantarum genera 4
Nucleic acid amplification test 366
Nucleus 31
Nutrition 36
Nystatin 105
O
Oatmeal agar 852
Ochratoxins 767
Ochroconis gallopavum 269, 271, 282, 283f
Ocular
candidiasis 407
Histoplasma syndrome 312
pythiosis 833
rhinosporidiosis 816
Oculomycosis 639
Oculosporidiosis 815, 816
Oidium 917
Ontogenic classification 34
Ontogeny 917
Onychocola canadensis 146, 176
Onychomadesis 917
Onychomycosis 146, 162, 175, 176f, 176t, 407
classification of 176
white superficial 177
Onygenaceae 313, 343, 715
Onygenales 299, 313, 343, 709, 715
Oogamy 830, 917
Oogonium 829, 917
Oomycetes 830t, 831
Oomycota 831
phylum 54
Oospore 831, 917
Ophiostoma stenoceras 230, 231
Opportunist fungi 11
Opportunistic mycoses 11, 399
Oral
candidiasis 405
ketoconazole 242
potassium iodide 607
Orellanine 772
Orellanus syndrome 772
Oropharyngeal thrush 408
Oropharynx 816f
Oryctolagus cuniculus 837
Osseous
blastomycosis 345
cryptococcosis 443, 444
Osteomyelitis 346, 405, 408
Ostiolate 917
Otitis externa 669
malignant 669
Otomycosis 669, 670, 671f, 671t
Ouchterlony's technique 871
Oxiconazole 109
P
Paecilomyces 640, 683, 687, 690692, 768, 783, 880, 918
lilacinus 691, 783
species 515, 640, 648, 683, 783
variotii 691, 692, 783
Pal's sunflower seed agar 451
Palmer surface, staining of 148f
Pancreas 499
Papilla 918
Paracoccidioides 379, 380, 713, 804
brasiliensis 36, 80, 300, 366, 379, 380, 380f, 381, 382, 385, 386f, 387f, 388, 389, 650, 791, 803, 871
lutzii 379, 381
species 309, 381, 613
Paracoccidioidoma 385
Paracoccidioidomycosis 379381, 383, 383t, 384
immunodiagnosis of 386
Paranasal sinus 741
mycosis 529
phaeohyphomycosis 275
Parasitophorous vacuole 498
Parenteral nutrition, total 409
Parenthesome 915
Parinaud's oculoglandular syndrome 384
Paris mushroom 770
Paronychia 407, 918
Paroxysmal nocturnal hemoglobinuria 833
Paspalum scrobiculatum 766
Patchy alopecia 167f
Pathogenic fungi 11
isolation of 872
Pectinate body 28, 180
Pedicellate sporangiola of Thamnostylum piriforme 575f
Penetrating keratoplasty 642f
Penicillate 918
phialides 692f, 693f, 783f
Penicillium 26, 27, 50, 507, 515, 640, 683, 687, 692, 723, 762764, 768, 782, 783, 851, 916, 918
aurantiogriseum 766
brevicompactum 690
camemberti 766
chrysogenum 12, 689, 690, 725
citrinum 690
commune 690, 766
decumbens 690
dupontii 690
expansum 768
funiculosum 690
glaucum 690
griseofulvum 106, 689, 766
marneffei 506, 507, 690
notatum 4
patulum 768
puberulum 765
purpurogenum 690
species 12, 34, 103, 640, 683, 690f, 782
verrucosum 768
Pentamidine isethionate 482
Peptone-yeast-glucose 839
Peptostreptococcus species 218
Pericarditis 408
Peridium 918
Periodic acid-Schiff stain 862
Perithecia 918
Perithecium 918
Perleche 408
Permanent immunodiffusion mount 871
Perosporomycetes 918
Petriellidium boydii 683
Phaeoacremonium parasiticum 282
Phaeoannellomyces 145
Phaeohyphomycosis 79f, 146, 251, 252, 269, 682
superficial 145
Phaeohyphomycotic cyst 270
Phaeoid 918
causative 146
fungal genera 257t
fungi 251, 258f, 644f, 780, 784
septate hyphae of 748f
hyphomycetes 640, 741
septate hyphae 785f
Phalloid syndrome 771
Phalloidin 771
Phalloin 771
Phenoloxidase test 850
Phenotypic switching 404
Phialemonium curvatum 270
Phialide 918
Phialoconidium 918
Phialophora 64, 257, 850, 918
parasitica 270
species 34, 258, 284
type 258
verrucosa 36, 251254, 258, 261f
PHOL stain 859
Phoma
conidia of 789f
hibernica 270, 789
minutella 270
sorghina 270
species 170, 788
Phospholipase 441
Phragmoconidia 918
Phycomycetes 811
Phylogenetic systematic 918
Phylogeny 918
Phyte 919
Phytone yeast-extract agar 713
Pichia anomala 614, 860
Piedra 154, 162
black 146, 154, 158, 159f
types of 154
Trichosporon species 157f
white 154, 155
Piedraia hortae 34, 48, 159, 159f, 160
Pigment, brick-red 514
Pimaricin 106
Pityriasis
amiantacea 169
versicolor 131, 131f, 132f
Pityrosporum orbiculare 128
Plant taxonomy 43
Plasmalemma 30, 31
Plasmogamy 32
Plectomycetes 53, 313, 343, 715
Pleistophora ronneafiei 496
Pleospora 789
Pleurococcus beigelii 154
Pleurostomophora richardsiae 44
Pneumocystis 473, 474, 478, 858, 863
carinii 4, 40, 473
strains 474
jirovecii 4, 8, 40, 43, 46, 473, 476, 477, 650, 866, 910
cysts, cluster of 480f
life cycle of 475, 476f
pneumonia 89, 443
taxonomy of 474
pneumonia 389, 473, 477
species 72
Pneumocystosis 473
PNS mycosis 146
Polyene 103
antibiotics 103, 104
Polyhexamethylene biguanide 499
Polymerase chain reaction 87, 719
Polyvinyl alcohol 859
Poroconidia 918
Potassium
hydroxide wet mount 856
iodide 112, 201, 242
saturated solution of 837
Potato dextrose agar 852, 878
Pradimicin 106
Prickly Acacia nilotica 298f
Prokaryotes 46, 47
Prophylaxis 455
primary 482
Propionibacterium acnes 218
Protein, major basic 739
Proteinase 421
Proteus vulgaris 218
Protista 797, 798, 829, 831
Prototheca 797799, 804806, 828, 854, 859, 913, 914
isolation medium 805
species 80, 363, 364, 798, 918
sporangiospores of 802f
yeast-like colonies of 803f
wickerhamii 797806
sporangiospores of 804f
zopfii 797, 798, 799, 800, 803, 804, 805, 806
sporangiospores of 804f
yeast-like colonies of 803f
Protothecal olecranon bursitis 800, 800f
Protothecosis 797, 798, 801
Protozoa 474, 494, 831
Proximal subungual onychomycosis 177
Pseudallescheria 683, 684
angusta 683
boydii 31, 39, 46, 48, 204, 209, 215, 217, 683685, 687
infection 108
species 79
Pseudoepitheliomatous hyperplasia 253
Pseudofungal infections 795
Pseudofungi 797
Pseudohyphae 26f, 76f, 78, 417t, 918
Pseudomonas 530
aeruginosa 218, 672
Pseudomycelium 918
Pseudomycetoma 210
Pseudosepta 787
Psophiacreptans 271
Psorospermiosis 811
Pulmonary
aspergillosis, chronic necrotizing 528
aspergillosis, semi-invasive 528
blastomycosis 345
candidiasis 405, 408
coccidioidomycosis 361
cryptococcosis 443
disease 526
chronic 345
histoplasmosis
acute 315
chronic 315
infiltrates 527, 724
mucormycosis 559, 560, 561f
pneumocystosis 478
residual lesions, chronic 361
Pulse therapy 138
Pulsed field gel electrophoresis 137
Purpureocillium 683, 687, 690692, 783
lilacinum See Paecilomyces lilacinus 44, 640, 691, 691f, 692f, 783
marquandii 691, 783
Pusillus 918
Pycnidia 918
Pycnidiospore 918
Pycnidium 28, 789, 918
Pyrenochaeta 217
mackinnonii 205
Pyrenomycetes 53, 890
Pyrenophora 787
Pyriform 918
Pythiaceae 831
Pythiales 831
Pythiosis 828, 829, 835t
arteritis 833
insidiosi 834
Pythium 797, 828, 829, 918, 920
aphanidermatum 828, 830, 833
gracile 829
insidiosum 26, 55, 77, 564, 828839
culture mount of 836f
growth of 836f
vaccines 838
wet mount of 835f
zoospores 870
species of Oomycetes 830f
Q
Quality management system 891
Quantamatrix multiplexed assay platform 92
Quercus acutissimia 697
R
Rabbit eye, infection of 284
Rachis 918
Racquet/Racket hypha 28, 28f, 180, 919
Raffinose 452
Ramichloridium mackenziei 270
Rasamsonia 44
Rattus
niditus 510
norvegicus 510
rattus 510
Refractive laser epithelial keratomileusis 642
Renispora flavissima 318
Replicate conidia 604
Respiratory disease, type of 346
Restriction fragment length polymorphism 538, 772
Reye's syndrome 766
Reynolds-Braude phenomenon 417
Rhexolytic cleavage 919
Rhinocerebral mucormycosis 559
Rhinocladiella 257
aquaspersa 252, 259, 261f
mackenziei 44, 270, 271
similis 259
species 284
type 258
Rhino-orbital mucormycosis 560f
Rhino-orbito-cerebral mucormycosis 560f
Rhinopoma hardwickei hardwickei 343
Rhinosinusitis, chronic 737
Rhinosporidioma 816
Rhinosporidiosis 811, 814
bony involvement of 817
pedunculated giant mass of 817f
polypoid mass of 815f, 816f
Rhinosporidium 811, 818, 918
kinealyi 811
seeberi 40, 78, 80, 303, 364, 651, 712, 797, 802, 804, 811814, 818, 828, 915, 919
ruptured sporangium of 812f
sporangia of 219f, 818f, 819f
sporangium of 819f
Rhizoids 571, 919
Rhizomucor
pusillus 556, 571, 575, 747
tauricus 556
Rhizomys
pruinosus 509
sinensis 506
species 509
sumatrensis 509
Rhizopus 556, 781, 880
arrhizus 556, 572f, 575, 740, 782f
homothallicus 572f
of mucormycetes 49f
microsporus 572f, 575, 782f
species 49f, 563, 571, 781
sporangia of 557f
Rhodotorula 854, 859, 914
glutinis 621
infections 614, 620
rubra 614
species 113, 621, 790
yeast-like colonies of 621f
Rhytidhysteron rufulum 271
Ribosomal vaccines 65
Rice starch agar 849
Rings within ring 171
Ringworm 162, 163, 166, 919
Rose Gardener's disease 229
Rose Handler's disease 229
Rose Thorn disease 229
S
Sabouraud's dextrose agar 556f, 621f, 685f, 692, 697f, 847, 784f
plate 535f, 836f
Saccharomyces
cerevisiae 12, 13, 474, 621, 790f, 860
infections 614, 621
species 12, 790
Saksenaea 558, 561, 563, 836, 853
erythrospora, white mycelial growth of 573f
loutrophoriformis 573f
oblongispora 572
species 556, 567
vasiformis 555, 573f
complex 571
Salicylic acid 151, 675
Salmonella typhimurium 517
San Joaquin valley fever 361
Sandal ringworm 174
Sanger's sequencing 90
Saprochaete capitata 619
Sapronosis 509, 919
Saprophyte 919
Sarocladium
kiliense 44, 693f, 694, 694f, 781
species 683
Scalp hairs 159
Scedosporiosis 684
Scedosporium 26, 271, 614, 640, 683, 684, 685, 687, 739, 880
apiospermum 31, 39, 110, 209, 217, 640, 683, 684, 685f, 686f, 834
aurantiacum 683
boydii 683, 684
dehoogii 683
inflatum 684
minutisporum 683
prolificans See Lomentospora 684
species 79, 113, 533, 683
Schenck's disease 229
Schizogony 36, 496, 498, 507
Schizomycosis 218
Schizophyllaceae 697
Schizophyllum commune 51, 683, 697, 697f, 762, 914
Schizosaccharomyces 915
pombe 474, 915
species 25, 507
Schmorl's melanin stain 865
Sclerotia 919
Sclerotic body 251, 253, 256f, 257f, 919
Sclerotic cells 253
Sclerotium 766, 919
Scopulariopsis 683, 690, 848
brevicaulis 164, 177, 692, 692f, 693f, 727, 783, 783f
brumptii 271, 683, 693f, 692, 727, 783
species 271, 683, 687, 783
Scopus 909
Scotch technique 77
Scotch test 385
Scutulum 919
Scytalidium
dimidiatum 149, 187, 920
hyalinum 187
lignicola 187
species 81, 696
Seborrheic dermatitis 133
Selenium sulfide 113
Semen culture 74
Sepedonium 781, 792
species 318, 792, 792f
Septate branching hyphae 279f
Septate fungal hyphae 673f, 746f
Septate hyaline hyphae 181f
Septate hyphae 30, 79f, 180f, 211f, 644f 645f, 746f, 747f, 784f
black-colored branching 645f
Septicemia 408
Septum 919
Serological tests 84, 241, 319, 367, 388, 871
Serology 83
Serum
precipitins 527
therapy 455
Setosphaeria 787
Sexual
reproduction 32, 919
sporulation 567
Shield cell 919
Sialic acids 441
Singapore ear 669
Sinobronchial allergic mycosis (SAM) 725
Sinus 206
mycetoma 739
opening of 206
Skin 74, 162, 499
involvement 453
lesions, keloid-like 301
prick test 725
test 85, 241, 367, 388, 422, 538
Society for Indian Human and Animal Mycologists (SIHAM) 904
Soil extract agar 853
Solid organ transplantation 362
Solitary adiaspiromycosis 711
Solitary lesion 445
Sordaria fimicola 13
Sotalia guianensis 300, 302
South American blastomycosis 379
Sphagnum moss 231
Spherical conidia 348f, 787f
Spherule 805, 912, 919
Spherulin 366
Spiral hyphae 28, 28f, 919
Splendore-Hoeppli phenomenon 347, 598, 601, 602f, 832
Splendore-Hoeppli reaction 919
Sporangia 50f, 798, 804f, 813, 819, 919
thick-walled 802f
Sporangiole 919
Sporangiophores 32, 49f, 557f, 573, 781, 919
Sporangium 32, 49f, 555, 557, 802, 919
Sporocyst 475
Sporodochium 919
Sporon 155
Sporophore 919
Sporoplasm 496
Sporothrix 201, 233, 240, 850, 919
brasiliensis 232
cyanescens 231
infection 232
luriei 241, 712
schenckii 14, 16, 46, 80, 229231, 240, 240f, 241, 241f, 284, 648, 791, 880, 914
complex 230
mycelial colonies of 240f
species 114, 229
Sporotrichin 242
Sporotrichosis 229, 231
Sporozoa 474, 812
Sporozoite 475
Sporulation
patterns, types of 258f
types of 240f
Squalene epoxidase 107
St. Anthony's fire, 764t, 767
Stachybotryotoxicosis 763
Staib's agar 451
Stains, differential 859
Staphylococcus aureus 218, 409, 741, 748
Stemphylium 785, 787, 789
solani 789
species 789
Stenella araguata 145, 146, 150
Sterigmata 919
Sterol quantitation method 885
Stramenopila 829, 831
Streptococcus
pneumoniae 59, 441, 857
species 218
Streptomyces 863
griseus 912
natalensis 106
nodosus 104
noursei 105
pimprina 102, 106
somaliensis 204, 205, 216, 217
species 28, 103, 209
viridis 205
Strongyloides 712
Subcutaneous
basidiobolomycosis 605
mycoses 11, 73, 146, 201
phaeohyphomycosis 272, 273f, 275f
protothecosis 800
of arm 800f
pythiosis 833
Sucrose 418, 452
Sulfamethoxazole 482
Sulfonamides 114
Sulfur 218, 219
Sunflower seed agar 850
Superoxide dismutase secretions 441
Swamp cancer 831, 834
Swimmer's ear 669
Swollen vesicle 569
Symbioses 920
Sympodial geniculate growth 280
Synanamorph 920
Syncephalastrum 781, 916
racemosum 574f, 556, 572
species 781
Syngamy 920
Synnemata 920
Synthetic antifungals 103, 106
Systema naturae 40
Systemic antifungal therapy 675
Systemic disseminated protothecosis 800
Systemic infections 155
Systemic lupus erythematosus 801
Systemic Malassezia infections 134
Systemic mycoses 11, 73, 146, 309
Syzygium cumini 697
T
Talaromyces 42, 506, 507
albobiverticillius 508
genera 515
infection 231
marneffei 25, 44, 81, 316, 506, 507, 508, 513f, 527f, 690, 717, 718, 870, 915, 920
cells 511
conidia 511
gene 516
infection 613
mycelial growth of 514f
purpurogenus 508
Talaromycosis 4, 9, 10, 36t, 42, 44t, 316, 320, 322t, 399, 506523, 614, 690, 715, 716, 920
Taxonomic status 31
Taxonomy 920
T-cell-mediated response, acquired 60
Terbinafine 107, 189
Terminalia cattappa 439
Tetrazolium reduction medium 853
Thallic conidial development 34
Thallus 920
Thamnostylum 573
lucknowense 573
piriforme 573, 575f
species 573
Thermophilic fungi 37
Thielavia subthermophila 271
Thiocarbamates 103, 106
Thymidylate synthetase 111
Tilletiopsis minor 51, 53t, 683
Tinea 146, 162, 166, 920
atypica 172
barbae 166, 172
blanca 154
capitis 163, 166, 167, 179f
corporis 166, 169, 170f
cruris 166, 173, 173f
double-edged 171, 171f
faciei 172, 172f
favosa 167
glabrata circinata 169
glabrosa 169
gladiatorum 171
imbricata 166, 171
circular lesions of 171f
incognito 172
indecisiva 171
inguinalis 173
manuum 166, 173, 174f
nigra 145, 146, 149f
palmaris 145
pedis 166, 174
profunda 169
pseudoimbricata 171
circular lesions of 171f
sycosis 172
unguium 166, 174, 176f
versicolor 131
Tioconazole 109
Tissue 74
section 279f
Tolnaftate 106
Torulopsis species 12
Toxic aleukia, alimentary 763, 769
Toxicum 770
Toxigenic fungi 11
Toxins 60, 404
Toxoplasma gondii 316, 514, 717
Trachipleistophora
anthropopthera 496
hominis 495, 496
Transmission electron microscopy 499
Transverse fission 513
Transverse septa 150f, 724f
Trebouxiophyceae 798
Trehalose 452, 804, 805
Trematosphaeria grisea 44, 204, 205, 216
Triacetin 113
Triazoles 109
Trichobacteriosis axillaris 157
Trichoderma 695f, 769, 784, 784f
citrinoviride 694
harzianum 694
koningii 694
longibrachiatum 694, 695, 784
polysporum 13
pseudokoningii 694
species 683, 783
whitish mycelial colonies of 784f
viride 694
Tricholoma magnivelare 772
Trichomycosis 920
Trichophyton 162, 163, 178, 188, 912
concentricum 165
gourvilii 164
mentagrophytes 28, 28f, 112, 164, 165, 169, 172174, 184f, 188, 671, 854, 868
rubrum 112, 165, 168, 169, 173, 174, 177, 183, 184f, 671, 854, 868
syndrome 178
schoenleinii 28, 162, 165, 167, 168, 179, 183, 185f
simii 164
soudanense 164
species 163, 177, 178, 182, 183, 183f, 189
tonsurans 28, 165, 168, 169, 183, 185f
verrucosum 169, 172, 185f
infection 168, 190
violaceum 28, 165, 167, 168, 172, 183
arthrospores of 168f
yaoundei 164
Trichosporoideae 155
Trichosporon 113, 133, 155, 158, 614, 615, 619t, 790, 854, 859, 913
asahii 133, 155, 157f, 615, 618
infections 614
asteroides 155, 615
beigelii 154, 155, 614
cutaneum 154, 155, 618, 619
infections 87
inkin 155, 615, 618
arthrospores of 618f
isolates 615
mucoides 155, 618
ovoides 155, 614, 618
species 32f, 51, 79f, 155, 155t, 156, 157, 160, 614, 615, 617f, 618t, 619, 696, 790
pseudohyphae of 616f
rectangular arthrospores of 158f, 617f
Trichosporonosis 154, 614
nodosa 154
Trichothecenes 768
Trichothecium 769
species 784
Trimethoprim 482
Trophocyte 813
Trophozoite 475
Trypanosoma cruzi 114
Tubulinosema acridophagus 495, 496
Tumor mass 206
Turkey-X-disease 765
Tursiops
aduncus 300, 302
truncatus 297, 300, 302
U
Ulocladium 785, 787, 789
chartarum 789
species 282, 789, 790f
Umbonate 920
Uncinocarpus 791
Undecylenic acid 113
Uniseriate phialides 535f
Unitunicate pyrenomycetes 270
Urease
medium 853
production 441
test 182
rapid 854
Urinary tract 405
candidiasis 407
Urine culture 74
Ustilaginomycotina 128
Ustilago
maydis 13
species 790
Ustomycetes 53
V
Vaccine 455
Vaginal secretion 75
Vaginal smears 621
Valley fever 356, 368
Vascular invasion 558
Vascular systemic pythiosis 833
Vasiform 920
Vegetative hyphae 27
Vegetative structures 27
Verrucose 920
Verticillate 920
Verticillium 920
species 784, 784f
Vesicles 535f, 920
Villose 920
conidia 604
Vinyl adhesive tape 77, 385
Visceral
cryptococcosis 443, 444
lesions 385
type 219
Vittaforma corneae 495, 496
Voriconazole 110
Vulvovaginitis 406, 408
W
Wangiella dermatitidis See Exophiala der matitidis 36, 281
Water insoluble
antifungal drugs 878t
drugs 877, 878
Water soluble 861
drugs 111, 878, 879t
list of 877t
Welsh's regimen 218
Wessely's ring 642, 642f
Westerdijk Fungal Biodiversity Institute 891
Wet mounts 75
Whitfield's ointment 112
White piedra 154, 155t
Wolff-Chaikoff effect 113
Wood pulp worker's disease 727
Wood trimmer's disease 561
Wood's lamp 179t, 920
examination 178, 179f
World Health Organization 773
Woronin bodies 31, 920
X
Xylohypha, See Cladophialophora bantiana
bantiana 280
emmonsia 280
Xylose 452
Y
Yeast 24, 25, 27, 52, 76f, 780, 789, 876, 920
black 27, 146, 147f, 257, 279, 281, 785
carbon base 853
cells 25f, 77f79f, 312, 363, 385, 386, 411, 411f, 413f
budding 26f, 448f, 449f
clusters of 136f
Gram-positive 410f
multiple 317f
of Candida species 411f
thick-walled oval 321f
extract phosphate agar 318
identification 417f
like fungi 640, 647, 741, 780, 789
multi-nucleated 347
nitrogen base 853
pasty colonies of black 147f
YeastOne Sensititre Assay 884
Z
Zearalenone 769
Ziehl-Neelsen staining 386
Zoonotic disease 3, 509
Zoophilic dermatophytes 164
Zoospore 831, 832, 920
induction of 870
Zoosporic fungus 831
Zygomycetes 55
Zygomycosis, See Mucormycosis Zygomycota 555
Zygomycotina 555
Zygospore 49, 49f, 555, 604, 606, 920
type of 555
Zymogen 30
Zymonema brasiliense 379
×
Chapter Notes

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General Topics in Medical Mycology
Chapters
  1. Introduction
  2. Fungal Morphology
  3. Fungal Taxonomy
  4. Immunity to Fungal Diseases
  5. Diagnosis of Fungal Diseases
  6. Antifungal Therapy
Medical Mycology is a newly established discipline of medical sciences, which has attained immense importance during the terminal two decades of 20th century, particularly after the onset of AIDS pandemic. In the past, fungi were believed to be merely non-pathogenic, commensals or contaminants, with exception of a very few pathogenic ones. But now these are recognized as medically significant organisms causing potentially life-threatening diseases invariably with fatal outcome. Therefore, this medical speciality is gradually gaining more and more importance in the present context, especially among the immunocompromised and debilitated patients with one or the other underlying risk factors.
This Section covers some of the fundamental issues pertaining to fungi, which are generally applied to the field of Medical Mycology. Their morphological features are entirely different from other causative agents like viruses, bacteria and parasites because they exist as yeast, mycelial or even both morphological forms. The understanding of these features is also essential to have high index of suspicion thereby timely detection of fungi as the causative agent in clinical setting of a particular disease.
The nomenclature, classification and taxonomy of many medically significant fungi are settled as and when teleomorphic state of a fungus is discovered, however, many of these agents are still included in phylum Deuteromycota i.e. ‘fungi imperfecti’ as no teleomorphic state is yet discovered. Other agents looking similar to fungi are now regarded as pseudofungal organisms like Prototheca, Rhinosporidium and Pythium species. Taxonomically such organisms are either classified as protistan parasites or oomycetes. These taxonomical intricacies are pertinent to understand the basic disease process adopted by a causative fungal agent.
How fungi overcome defense mechanisms of human body among immunocompetent as well as immunocompromised hosts is separately dealt in one chapter of this Section. By the time fungal infection is recognized, patient is found to be already immunocompromised and remedial modalities become quite cumbersome thereby such infections invariably prove to be fatal. From prophylactic point of view, role of vaccination in Medical Mycology is still in its rudimentary stage. Hence general hygienic measures are recommended for prevention of fungal diseases.
A high index of suspicion is essential in any clinical setting for diagnosing fungal diseases, which is often lacking among medical specialists. The diagnostic techniques are utilized to prove or disprove the suspicion thereby therapeutic modalities are resorted, accordingly. Therefore, an appropriate diagnostic approach to fungal diseases is also described briefly in separate Chapter of this Section.
After establishing the final diagnosis, appropriate antifungals are to be instituted the therapeutic modality. However, some of the fungi, yeasts as well as mycelia, are developing resistance to the commonly used antifungals. It may be because of the indiscriminate use of such drugs merely on an empirical basis, without specific indication and/or appropriate antifungal susceptibility testing. The understanding of antifungals is of paramount importance thus commonly used antifungals have been described.
The diagnostic modalities and antifungal susceptibility testing are important components of the management of fungal diseases. Hence all the technical details pertaining to such issues are separately given in the Section as Appendices.2

3IntroductionCHAPTER 1

The fungi are achlorophyllous eukaryotic organisms, which multiply sexually and asexually by production of spores. Their somatic structure is composed of either yeast or filament bordered by cell wall, which is mainly composed of chitin. The fungi, unlike plants, cannot produce their own food thus are called heterotrophs. It was generally believed in the past that fungi were plants and classified under kingdom Plantae. However, now these are classified under independent biological kingdom i.e. Fungi since 1969.
The fungus is essentially a Latin word that means mushroom. The branch of biological science, which deals with study of fungi, is known as Mycology (Myco+logy=- Fungus+study, Gr. Mykes, mushroom/fungus, logos, discourse/study). The term is basically derived from mykes, Greek word, which is also used for mushroom. Hence Medical Mycology is the study of epidemiology, ecology, pathogenesis, diagnosis and therapeutic modalities of fungal diseases found prevalent among human beings. This discipline of Medical Microbiology is emerging rapidly and has now attained full-fledged and independent status in biomedical fields. The subject of mycology is essentially contemplation of medical sciences in newer perspectives.
Analogous to the field of Medical Mycology, Veterinary Mycology is study of fungal infections among animals, which often presents in similar way as those found in human beings. Sometimes, these are transmitted from animal-to-human beings and such infections are called zoonotic diseases. In recent reports cats are recognised as source of human sporotrichosis in Brazil establishing animal-to-man transmission. Hence reference of animal mycoses in the Textbook of Medical Mycology is essential to understand the epidemiology of a fungal disease.
The diseases of warm-blooded animals caused by fungi are known as mycoses (singular = mycosis). The prefix ‘myco’ has been given to few bacterial organisms like Mycobacterium and Mycoplasma or diseases like Mycosis fungoides i.e. cutaneous T-cell lymphoma, etc. that were speculated in the past to have similarities with either fungi or diseases caused by them. But later on it was realized that these organisms are not at all related to fungi. However, their nomenclature is still popular as misnomer because these are now well-accepted terms of the organisms and/or diseases.
 
Historical Perspective
The clinical manifestations of some of fungal infections are known since antiquity, however, systematic study of fungi is hardly one and half century old. In 1835, Agostino Bassi (1773-1856) in Italy established that fungus (Beauveria bassiana) was the cause of disease (muscardine) in silkworms (Bombyx mori), which could be transferred from one silkworm to the other. This fungus has shown to be widespread geographically and is currently used as a biological agent for the control of important insect pests in agriculture. Bassi is rightly acknowledged to be the first to refer the etiology of an animal disease to a microbial infection and is universally regarded as “Father of Mycology”. On the basis of these findings he predicted that fungi could also cause infections in man.
In 1842, David Gruby demonstrated for the first time that infection of scalp (favus) was caused by fungus through its inoculation to the healthy skin. Gruby thereby fulfilled what later on became famous as Koch's postulates, comprising the criteria necessary for the acceptance of a microbe as the cause of a specific infectious disease. Since then knowledge of fungal infections, which was fragmentary to begin with, has increased exponentially over the years as an independent infectious disease speciality as Medical Mycology.
The discipline of Medical Mycology attained substantial recognition in the field of bioscience in 1910 when 4French dermatologist and microbiologist, Raymond Jacques Adrien Sabouraud (1864-1938), published his seminal treatise, ‘Les Teignes’. This monumental work was a comprehensive account of most of the known dermatophytes, which is still being referred by the mycologists. He laid down solid foundation of the field of Medical Mycology, which gained remarkable momentum. As a result official journal of ISHAM was initially named as Sabouraudia in his honour in 1961. The medium for cultivation of fungi still bears his name, which is universally used for primary isolation of pathogenic and non-pathogenic fungi from clinical as well as non-clinical specimens in the mycology laboratory. It is suitable for the growth of both yeasts as well as mycelial fungi. Therefore, Raymond Sabouraud has rightly been called “Father of Medical Mycology”.
Similarly, P A Saccardo has played significant role in the establishment of field of Medical Mycology in earlier days. The other scientists who have played an active role in development of mycology are J I Schoenlein, Norman Conant, Chester Emmons, David Gruby, H P R Seeliger, J W Rippon, L Ajello, K J Kwon-Chung and Arvind A Padhye.
The introduction of fungi to man came into existence through the findings of Agostino Bassi in 1835 and the field of Medical Mycology has now completed more than 1½ century (mid-19th and 20th) as it has now entered into the 21st century. There have been lot of developments during this period, specially after advent of modern molecular techniques, which are dealing with diagnostic as well as taxonomical matters. But many issues are still lying unresolved before the mycologists, which include classification of pseudofungal organism and successful cultivation of Lacazia loboi and Pneumocystis jirovecii on artificial culture media. It is expected that molecular techniques will be able to resolve most of the intricacies of pending issues in the field of medical mycology in the times to come.
There are significant developments in treatment modalities of fungal infections and we have now achieved new prospects. However, till 1950s there was no specific antifungal agent available. Nystatin was discovered in 1951 and subsequently amphotericin B was introduced in 1957 and was sanctioned for use for human beings. In 1970s field was dominated by azole derivatives. Now, this is the most active field of interest, where potential drugs are being developed to treat fungal infections. By the end of 20th century, fungi have been reported to be developing drug resistance, among yeasts and mycelia in hospital as well as community acquired infections.
 
Milestones in Medical Mycology
The discovery of relationship of certain fungi to disease precedes work of even Louis Pasteur and Robert Koch. Some of the important milestones in Medical Mycology are given below:
1835
Bassi described fungal etiology of ‘muscardine of silkworms’.
1839
Schoenlein studied fungal infection of scalp i.e. tinea capitis (favus). Remak, succeeded in growing the fungus on apples and reproduced the disease in animals and on his own forearm.
1839
Lagenbeck described yeast-like organism of thrush.
1842
Gruby independently isolated fungus on artificial media responsible for tinea capitis and produced disease by inoculation of healthy scalp.
1890
Sabouraud began publishing large numbers of articles on fungal disorders of skin, which eventually culminated in an enormous contribution to the field of Medical Mycology.
1928
Penicillin was discovered by Alexander Fleming as product from Penicillium notatum (P.chrysogenum).
1934
According to Botanical Nomenclature, species concepts of dermatophytes was redefined by Chester Emmons.
1969
Separate Kingdom - Fungi, was created by Whittaker, in the five-kingdom classification.
1976
Organism causing human PCP i.e. Pneumocystis carinii was proposed as P.jirovecii by J K Frenkel, in honour of Czech Parasitologist Otto Jirovec, who described this microbe in humans but he is not widely recognised.
1999
Pneumocystis carinii strain infecting human was re-designated as P.jirovecii.
2002
Loboa loboi was re-designated as Lacazia loboi and C.posadasii was established as new species of genus Coccidioides.
2017
Penicilliosis Marneffei was renamed as Talaromycosis and Emmonsiosis as Emergomycosis.
The observations of both Bassi, in 1835 and Gruby in 1842, demonstrating that fungi could be cause of diseases, was long before Pasteur's Germ Theory came into existence fulfilling Koch's Postulates, about four decades before Koch actually formulated them in 1884.
The start of the modern age of mycology begins with Pier Antonio Micheli's (1679-1737) publication of Nova Plantarum Genera. Published in Florence, this seminal work laid the foundation of the systematic classification of 5grasses, mosses and fungi. He is considered as the founding ‘Father of Scientific Mycology”.
Invasive fungal infections are becoming an increasingly important cause of morbidity and mortality, particularly for immunocompromised human populations. The fungal pathogens belonging to genera Candida, Cryptococcus and Aspergillus collectively contribute to over 1 million human deaths annually.
 
Epidemiology
The fungi are widely found in environment and most of them are harmless commensals, contaminants or non-pathogenic agents. Some of the fungi are even useful to mankind in several ways. However, small number of these organisms are causing disease among men, animals and plants. This has been estimated by Hawksworth that only 5% of total fungal species in world have been identified which constitute about 70,000 species out of an estimated 1,500,000 but hardly 600 are usually recognized as primary pathogens of man and other mammals. Out of these fungal species, less than 100 are frequently encountered in routine clinical practice. It is estimated that about 1,500 new species of fungal genera are being described every year.
By 1995, approximately 70,000 fungal species have been accepted as compared to 5,000 viruses and 3,100 known bacteria. Yet remarkably, very few of these fungi cause disease in humans. Moreover, most of fungi exist as molds but there are number of pathogenic yeasts and some of them are dimorphic as well. The dimorphic fungi exist either as yeast or spherule in their parasitic form when causing infection to host and assume mold form when growing as saprotrophic in nature. Out of 1.5 million species of fungi estimated to exist all over the world and one-third of global fungal diversity found in India. According to Maheshwari there are 27,000 species of fungi, which have been recorded from India.
As fungal infections are not notifiable like viral, bacterial or parasitic diseases hence these are not given much attention and usually diagnosis is established very late when the patient is terminally sick. The approach to identify fungi in developing countries is on gross morphological features whereas in developed countries it is molecular based approach that is important due to availability of requisite infrastructure. Moreover, most of serious fungal infections are more common in developed countries due to underlying immunocompromised situations as compared to developing countries where majority of disease are due to low hygienic standards and various environmental factors.
The overall incidence and prevalence of mycotic infections is increasing, particularly during the last three decades. A major contributor to this emergence is growing number of immunocompromised and more susceptible individuals. Previously the epidemiological features of these diseases were not well-documented but the understanding of epidemiology of fungal infections has increased considerably in recent years, largely because of studies that have mainly focused on specific patient groups, hospital series and autopsy surveys.
Candida species is the fourth most common organism recovered from blood cultures in hospitalized patients. Aspergillosis is common in selected populations, such as bone marrow transplant recipients and lung is the most common clinical site. Aspergillus species is isolated in substantial number of solid organ and bone marrow transplant recipients with clinical findings of pneumonia. In such patients, high mortality rates are observed in relation to Aspergillus species.
The diseases, which were not prevalent in a particular area, are now being reported very commonly due to frequent travel across the globe. In the times to come, diseases like paracoccidioidomycosis and coccidioidomycosis which are currently found in restricted zones of endemicity in the New World, may be reported from other parts of world. Some of fungal infections are only limited to African continent like Histoplasma capsulatum var. duboisii. Blastomycosis is also found in Africa in addition to its native prevalence in the southeastern parts of North America.
The liberalization and globalization of economy has increased the gap between rich and poor as well as urban and rural population. Consequently privatization of health services has targeted poorest of poor due to migration of laborers thereby affecting epidemiology of fungal diseases.
The fungi are now recognized as significant cause of morbidity and mortality among man and animal. They have emerged as important etiological agents of opportunistic infections as well as full-fledged diseases as true pathogens. The invasive fungal infections were regarded as very rare till half century ago. Since that time there has been steady increase in number of patients suffering from life-threatening fungal infections, specially in prevailing immunocompromised circumstances. In recent 6years opportunistic fungal infections have emerged as one of interesting areas due to number of contributory factors. This calls for increased awareness about fungal diseases and their definitive diagnosis. Care must be taken to exclude such type of infection in cases where fungus is grown in culture but not seen in lesion by smear preparations and histopathological examination of tissue sections.
Like other organisms, Koch's postulates are applicable to fungal infections as well despite the fact that some of agents like Lacazia loboi are yet to be successfully grown on artificial culture media. The present challenges in Medical Mycology are diagnosis, treatment, taxonomy, mycology training and changes in diseases patterns. However, new and more effective tumour chemotherapeutics, methods of surgery and irradiation, complex treatment procedures such as autologous and allogenic bone marrow and blood stem cell therapy as well as organ transplantation, have enabled treatment of various solid tumors and systemic hematologic diseases. The use of such chemotherapeutics and immunosuppressants as therapeutic modalities result in general increase in opportunistic bacterial, viral parasitic as well as fungal infections. The epidemiological features of important fungi are dealt with subsequent chapters of this Textbook.
As such, the arthropods are not acting as biological vectors of the fungal diseases however; sometimes these may act as mechanical carriers of fungi. In this regard, cockroaches have been reported to the source of transmission of fungi in hospital set-up.
As per the statement of G C Ainsworth in 1966, that in reality it is the distribution of the medical mycologists or active investigators in this field and not the true distribution of fungal disease, which is reflected as its epidemiology.
 
Ecology of Fungi
The fungi are capable of existing and flourishing in a wide variety of environment as parasites, saprotrophic or symbionts. In the past they were known to be cave dwellers but presently they are found existing on innumerable places. They may be parasites of organic substrate, such as wood and other decaying plant parts, paper, leather, cloth, keratinous and chitinous substrate, oils and fats, resins and even petroleum and draw their nutrition.
The definite ecological groups of fungi known to exist as soil fungi, aquatic fungi, coprophilous fungi (grow on animal excreta), entomogenous fungi (parasites of insects) predacious fungi (capture small animals and protozoa), marine fungi, osmophilic fungi (from saline soil) and thermophilic fungi in which organism show remarkable adaptations not only to exploit nutritionally rich substrate but also to reproduce themselves. As decomposers, fungi are essential because along with bacteria, they recycle vital elements, such as nitrogen and phosphorus, back to the ecosystem.
Although beneficial effects of fungi far outweigh their harmful aspects but there are some disease-causing and destructive species, which are clinically significant. The number of saprotrophic fungal species far outnumbers those that are parasitic and impact on the health of man, animal and plants. This is well-known that nearly one hundred saprotrophic fungi, which may have their existence in any of common habitats, mentioned above can adapt to infect man and animals and cause diseases. It is also essential to acquire sound knowledge of biology of these fungi and their role in ecological niche. The limited data is available about survival of fungi that commonly cause health-care associated infections in immunocompromised patients on typical hospital materials. This indicates that many of fungi like Candida, Aspergillus and Fusarium species, which are responsible for health-care associated infections, survive for at least one day and often longer on fabrics and plastics routinely used in hospitals. These survival results indicate fabrics and plastics to serve as potential reservoirs or vectors for fungi.
There have been three ecological categories of infectious mycological agents based on their natural habitat. Therefore, traditionally fungi have been described as geophilic, zoophilic and anthropophilic. Now, a fourth category has also come into existence that is called hydrophilic, which encompasses free living organisms that live in aquatic habitats on non-living organic matters or on aquatic plants. The diseases, caused by such fungal (lacaziosis) and pseudofungal organisms (rhinosporidiosis and pythiosis) are currently categorized as hydrophilic infections.
 
Magnitude of the Problem
Mycology is a subject matter of immense interest to botanists as most of fungi are plant pathogens. The veterinarians also take lot of interest as fungi are significantly affecting health of domestic animals as well. Now, those fungi, which were supposed to be non-pathogenic, are reported significantly infective agents. Hence microbiologists and histopathologists are taking more interest in this branch 7to render diagnostic services in hospitals. The field of Medical Mycology that was virtually limited to case reports in the 19th century had attained status of full-fledged subject, as an important branch of medical science by the turn of the 20th century. It is estimated that during the 21st century, magnitude of problem is going to be increased because of the immunocompromised nature of hosts entailing in secondary as well as primary fungal infections.
Another way to estimate that this branch has expanded enormously in recent past, is by increased number of publications on issues related to Medical Mycology. Now, not only case reports but detailed prospective and retrospective studies are being published regularly in national and international journals, more in number and quality to previous times and as such subject of Medical Mycology has entirely changed over the years. Therefore, the field of Medical Mycology has evolved to the present independent status as significant branch of medical sciences.
Although fungi were recognized as disease causing agents much earlier but their significance was overshadowed by bacteriology and even virology. The attitude towards Medical Mycology was ‘step-child’ of our doctrine and research. It was least bothered subject in medical institutions as compared to other branches of medical sciences. But now there is ‘obligatory’ attention to this subject because clinicians face challenges due to underlying immunocompromised situations leading to life-threatening secondary fungal infections taking lives of innumerable patients. The fungal infections had such an impact that now-a-days no health personnel can ignore mycology, no matter whatsoever may be their speciality.
In the past, Mycology Sections of medical institutions, used to get only few skin scrapings from the Department of Dermatology and Venereology. In due course of time this field has evolved to an extent that Mycology Sections are now receiving clinical specimens from almost all specialties whether medical or surgical. Moreover, their number and quality has enormously increased in recent past due to greater awareness about fungi as disease causing agents and microbiology laboratories are now receiving more number of clinical specimens for mycological investigations. Therefore, due to the circumstantial compulsions, the microbiologists and pathologists have left their myopic attitude towards the fungal infections.
As fungal diseases are not notifiable diseases hence data on morbidity and mortality attributable to these diseases undoubtedly is understated. But incidence and prevalence of fungal infections has increased enormously. It may be due to underlying predisposing factors such as immunocompromised situations like use of corticosteroids, antimicrobials, immunosuppressive and anticancer drugs, bone marrow or solid organ transplants, HIV-positivity, metabolic disorders i.e. diabetes mellitus, etc as narrated in Table 1.1. Out of all these factors two are most important and prominent namely therapy among cancer patients and pandemic of the AIDS. Moreover, elderly patients, whose life span has been extended by treatment of cancer or other debilitating diseases, are more susceptible to secondary fungal infections than younger individuals. In addition to that there are changing parameters of mycoses like host population, emerging drug resistance and reporting of newer fungal pathogens.
Table 1.1   Predisposing Factors of Fungal Diseases.
  • Widespread use of broad-spectrum antibiotics
  • Prolonged use of steroids for more than three weeks
  • Increased number of intravenous drug abusers
  • Greater number of immunocompromised patients
  • Increased number of AIDS cases with longer survival
  • More aggressive therapeutic modalities of cancers
  • More intervention in intensive care medicine in adults
  • Catheter-borne infections among patients
  • New and more widely used prosthetic devices
  • Increased number of BMT and SOT procedures
The rapid growth of fungi such as Candida albicans in debilitated patient with non-mycotic disease may be minor factor as cause of death or in contrast the other fungal infections. Similarly, lack of experience in diagnostic techniques, inadequate laboratory infrastructure and lack of pathological autopsy examination, may limit recognition and reporting of many deaths due to fungal infections. These days, it is feasible to detect about 30% of diagnosis of fungal infections are made antemortem and rest 70% as postmortem. This paradigm is going to be shifted depending on the awareness among the medical personnel.
The basic need of hour is to create awareness among clinicians so that they are motivated to an extent as to send samples to Mycology Section of Microbiology Department. In most of advanced medical institutions, it is estimated that about five percent autopsied patients turn out to have 8died of fungal infections. At present in most of the teaching hospitals every third clinicopathological conference (CPC) is reporting deep-seated fungal infection as secondary invader responsible for terminal events of deceased patients. The autopsy findings also substantiate an increasing prevalence of invasive fungal infections among hospitalized patients along with significant changes in aetiology and underlying disease process. Therefore, pathological autopsy is an essential feedback for clinicians in particular setting of severely immunocompromised population and emphasizes constant and urgent need for more efforts towards prevention, diagnosis and treatment of fungal infections. The autopsy besides being diagnostic method, is also a means of medical quality control because it acts as deterrent on the diagnostic procedures.
The prognosis among patients of fungal infections as such is very poor. Therefore application of early diagnostic and therapeutic modalities is essential. A fruitful outcome depends on a very close association between Medical Mycology laboratory and clinician incharge of concerned specialty. It has also been observed that increased awareness among medical personnel has contributed to an early diagnosis and commencement of prompt and timely treatment in life-threatening fungal infections. Despite all the available modalities, Medical Mycology is still labeled as tip of the iceberg as little is known about it to the medical fraternity.
The radiological investigations, like ultrasonography, computed tomography, magnetic resonance imaging, have contributed significantly, in addition to routine x-rays examinations. Now, there are effective chemotherapeutic agents available and further modalities are being developed for treatment of fungal infections, therefore, their application requires proper techniques. The opportunistic fungal infections vary greatly in their clinical and pathological manifestations due to underlying illness of patients at risk as well as treatment modalities undertaken.
The newly isolated fungi may be morphologically atypical because of partial treatment given to patient. Therefore, if fungus does not fit into any description provided, serial transfers on Sabouraud dextrose agar may be necessary to restore typical morphology. When fungal growth is observed from clinical specimen, three principal characteristics to be evaluated are texture, growth rate and pigmentation. Therefore, using these criteria, conventional fungal identity can be narrowed down quickly to a limited group of fungi having similar characteristics.
 
Burden of Fungal Diseases
In a large number of countries two organizations i.e. Leading International Fungal Education (LIFE) and Global Action Fund for Fungal Infections (GAFFI) have conducted extensive campaign to document the burden of fungal diseases in respect to that country. This data has been published in various medical journals and cited in Further Reading of this Chapter as well.
 
Fungal Taxonomy
During the 20th Century, until around 1950s, botanists used the term fungi to include all members of ‘plant kingdom’ that did not have stems, roots, leaves and chlorophyll. By this definition, even some bacteria were included with fungal category. As fungi were initially classified with plants hence lot of botanical influence is seen on this subject. This may probably be the reason that Medical Mycology has been mainly subject of interest to the botanists. These organisms were transferred to an independent kingdom, after five-kingdom classification by Whittaker in 1969, based on their cell morphology that subsequently substantiated by molecular studies. Now, after recent increase in prevalence of fungal infections, especially in immunocompromised patients, it has become subject matter of great interest to microbiologists, pathologists, internists and the allied medical specialists, who regularly encounter such type of infections.
The Medical Mycologists encounter many new fungal genera and species that are known to have clinical relevance and every year many species are being added in the list of significant fungal pathogens. Many fungi, formerly considered as insignificant commensals or merely laboratory contaminants, have now taken new roles as agents of infectious diseases in patients with AIDS and other immunodeficiency conditions, in drug abusers and in debilitated individuals with various underlying diseases. The subject has reached a level of understanding where any fungus recovered as pure culture from any of the body sites must be considered as potential human pathogen.
In recent times, the human genome has been fully described and this knowledge, based on molecular techniques, is applicable to Medical Mycology as well so that diagnostic dilemma and taxonomic confusions are now feasible to be resolved. In this very sequence a long-standing taxonomical controversy of Pneumocystis jirovecii has been substantially resolved. Microsporidium species had a similar fate. These atypical fungi are shifted from kingdom 9Protista to Fungi; thereby Parasitology to Mycology; hence being described in the Textbook of Medical Mycology.
The pseudofungal organisms like Pythium and Prototheca species are mycelial or unicellular microorganisms that are not classified in the kingdom Fungi and that produce infections with clinical and histopathological features resembling those caused by eumycetes. Taxonomically, on the basis of molecular studies, Rhinosporidium seeberi is also now considered as a protist, classified in Mesomycetozoea, recognized as DRIPs clade of protistan parasites.
The Medical Mycology is totally new and unfamiliar subject to medical personnel and presents difficulties, which are apparent while identifying newly isolated fungal agent. Even though procedures for direct examination and isolation have been mastered, mycologist is often left to confront fungal culture that may be totally unfamiliar both macroscopically and microscopically. This happens most frequently when person is new to this field or knows least what to do. In such a situation, even if the species identification is difficult, at least the isolate should be identified to the genus level and immediately conveyed to the clinician so that timely and definitive treatment is instituted then and there. In due course of time, further taxonomical studies can be performed for final identification of the clinical fungal strain.
 
Endemic Mycoses
The endemicity of some of the fungal diseases like histoplasmosis, blastomycosis, coccidioidomycosis, paracoccidioidomycosis as well as talaromycosis are restricted to limited geographical areas. This may be due to environmental and other factors, which favor growth of fungi in soil of these areas. Histoplasmosis and blastomycosis mostly afflict people in Mississippi and Ohio River Valleys, while coccidioidomycosis is found primarily in southwest desert regions of USA and paracoccidioidomycosis in certain parts of Latin America. However, talaromycosis is found prevalent in entirely different geographical zone away from Americas i.e. Southeast Asia. All these diseases are acquired through inhalation of air contaminated with conidia of mycelial phase and cause localized to diffuse pulmonary involvement entailing dissemination to distant body sites. Their clinical features are different but all of these fungi are dimorphic in nature.
There are now case reports of endemic mycoses from non-endemic areas also. In case of coccidioidomycosis, new area in northeast Brazil has been recognized. In this very context talaromycosis has been reported from Africa without patient's history of travel to its endemic zone. In India, it has been now significantly reported from Manipur, Mizoram, Nagaland and upper Assam. It may be because of its higher prevalence in adjoining countries like Myanmar and northern Thailand.
 
Imported Mycoses
Those fungal infections which are caused by endemically prevalent fungi, encountered in non-native locations are called imported mycoses. In this era of modern technology, the entire world has become a ‘global village’ and borders of diseases are being demolished day-by-day. Now, due to frequent traveling all over the world in this era of jet age and underlying immunocompromised situations, zones of endemicity are not limited to their original native areas of prevalence. Such types of endemic fungal infections are also on the rise.
 
Emerging and Re-emerging Fungal Diseases
In the past fungi were considered to be merely non-pathogenic or simply laboratory contaminants with very few exceptions with cutaneous manifestations. But due to circumstantial immunocompromised background among the patients, these very non-pathogenic and contaminants have now proved to be significant pathogens and are encountered as emerging agents of life-threatening fungal diseases. In addition, some of the clinical entities which were not of great magnitude have recently re-emerged as important diseases. Most of the dimorphic fungi have re-emerged after the advent of AIDS and other immunocompromised situations.
In Medical Mycology as such there is an emergence and re-emergence of different types of fungal diseases of paramount significance, which are caused by Saccharomyces cerevisiae, Candida auris, Scedosporium apiospermum, Talaromyces marneffei and Fusarium species, which have recently been reported in literature. Outbreaks due to fungal infections do occur but only in sporadic form and epidemics have not been reported. For example, incidence of cryptococcal meningitis has recently increased to 1000-fold in the New York City alone.
In addition, during late September 2012, a multistate outbreak of CNS fungal infection and septic arthritis was caused due to an emerging fungus, Exserohilum rostratum in the USA, which is otherwise a plant pathogen. There 10were over 700 patients with 64 deaths, who received epidural injections of methylprednisolone produced at a Massachusetts compounding center and subsequently developed meningitis with or without posterior circulation stroke and/or spinal or para-spinal infection and more than 30 patients who received intra-articular injections of the same drug developed osteoarticular infections.
Some of the fungi, which have been reported as merely environmental contaminants, have now emerged as significant human pathogens. Some of mucormycetes like Apophysomyces variabilis, Saksenaea erythrospora and Rhizopus homothallicus have now emerged as significant cause of various forms of mucormycosis and if not timely treated may lead to fatal consequences even in immunocompetent individuals.
 
Medical Mycology and AIDS
Mycology as such was an unknown field of medical sciences except in some of clinical specialties like Dermatology and allied subjects dealing predominantly with dermatophytosis and candidiasis. The real augment came three and half decades ago when new virus was brought to the human civilization probably a spillover of biological warfare laboratories, which created havoc by abrogating immune system of its targeted victims. Consequently the index case was recognized in the USA in 1981. The novel agent was subsequently designated as Human Immunodeficiency Virus (HIV) in 1986, the labeled cause of Acquired Immunodeficiency Syndrome (AIDS).
This type of clinical setting gave an open invitation to all the secondary invaders of viral, bacterial, parasitic and even fungal origin. The patients virtually became microbiological culture plates and all these organisms thrived upon them. The fungi were already well-known agents invading individuals with weaker immune system, made their abode among AIDS patients thereby leading to fatal consequences. This duo of HIV and fungi attained medical acceptability to an extent that some of the diseases like pneumocystosis, cryptococcosis, talaromycosis, etc. became AIDS-defining illnesses. Therefore, the fungal disease/parameters started defining the viral infections for the first time in the history of medical sciences.
Now, every year fungal pathogens cause more than two million infections leading to high morbidity as well as mortality. Out of this two fungal genera i.e. Candida and Cryptococcus are together accounting for about 1.4 million infections with higher mortality rates. The patients with disturbed immune system like HIV/AIDS, are mainly vulnerable to such infections. Although there is substantial control on this disease due to introduction of ART but fungal infections are still on the higher rates.
Hence advent of AIDS has altered the incidence as well as prevalence of fungal infections thereby the course of action of the field of medical mycology. During this period since 1981, epidemiological scenario about fungal infections has entirely changed. The fungal isolates, which used to be discarded considering as merely laboratory contaminants are now found to be playing significant role in pathogenicity of the variety of infections. This is true with fungi affecting not only immunocompromised patients but about those capable of infecting even healthy individuals as well. Therefore, the impact of syndrome is such that even true fungal pathogen like Histoplasma, Blastomyces, Coccidioides and Paracoccidioides species, infect immunocompetent individuals as well as now immunocompromised patients also and that too in a very large number.
This was probably warfare experiment to begin with but in due course of time it has attained the level of a full-fledged pandemic as it is now reality and not fiction-based story of a syndrome of ‘sealed’ origin. In the times to come, fungal infections will be encountered more in number, killing thousands of patients with underlying abrogated immune system.
 
Biological Warfare and Fungi
During the events of nine-eleven (September 11, 2001), so-called greatest imperialist power of the world was 'shaken' to the hilt. This was followed by incidents of spreading white powdery material pertaining to Bacillus anthracis. All these developments led to apprehension that fungi also might be used on similar pattern. Most of the governments secretly resort on to biological warfare acts for their survival in power through state organized terrorism thereby giving rise to origin of counterproductive terrorist groups, who may retaliate by indulging in such activities.
As such till date no fungal agent has been apparently used in biological warfare but reports are available about their secondary metabolic products. In the past, Yellow Rain in Laos is one of examples, where mycotoxins (Trichothecene) were used as agent of biological warfare. Similarly, Agent Orange, a herbicide and defoliant was also used during Vietnam War. The degradation of 11such material release dioxins, which cause serious health related maladies.
The danger of working with cultures of some of fungi like Coccidioides species, were even recognized as early as in 20th century. Now, in the changed international scenario, there is an apprehension that these fungi may be abused as biological warfare agents. Therefore, their interstate transport is restricted under provisions of US Anti-terrorism and Effective Death Penalty Act, 1996. These highly infectious agents require safe laboratory handling under Biosafety Level 3 facility.
 
Classification of Fungal Diseases
Most of the fungi are saprotrophic in nature as they use non-living organic material as source of their nutrition. They are significant scavengers in the ecosystems. Along with bacteria, they are important in recycling carbon, nitrogen and essential mineral nutrients. As far as their interaction with living world is concerned, they have various modes of existence like other microorganisms. The host-parasite relationship in fungi can be divided into three potential modes namely mutualism, commensalism and parasitism depending upon the basic interaction between fungus involved and the host. These are as follows:
(a) Commensalism: The fungus neither gets benefit nor harmed in the host-parasite relationship. There is usually no physiological interaction between two organisms. One organism uses other to get a better position in the environment.
(b) Mutualism: The fungus and host take mutual benefits from host-parasite relationship. For example in Mycorrhizae there is association of fungi with plant roots and in Lichens there is association with algae and cyanobacteria.
(c) Parasitism: The fungus gets benefits and host is harmed by host-parasite relationship. Most of the fungi causing disease in humans, animals and plants are placed in this category.
The basic mechanism of fungal pathogenicity is its ability to adapt to tissue environment, physiological barriers and temperature (37°C) thereby withstand lytic activity of hosts’ cellular defense mechanism. The same fungal agent can produce different manifestations depending upon underlying situations of the host. Based on their wide-spectrum of adaptability, various fungi causing human mycoses can be categorized into following groups:
(a) Pathogenic Fungi: These fungi have ability to adapt to the tissue environment, which is quite marked. This is expressed as thermal dimorphism and fungi are able to cause infection even in the immunocompetent individuals.
(b) Opportunist Fungi: These fungi cause infections only in patients with immunodeficiency or other debilitating conditions, which carry high morbidity and mortality. Some sort of defect in immune system of the host is prerequisite for establishment of such fungal infection and these do not exhibit thermal dimorphism.
(c) Toxigenic Fungi: These fungal species cause illness or even death of patients and/or animals after ingestion of contaminated food by fungi (mycetismus) or their mycotoxin metabolites (mycotoxicosis).
(d) Allergenic Fungi: These fungal species act as allergens and cause various allergic manifestations in human beings.
The disease process is established on the basis of net outcome of host-parasite relationship. In addition, the fungal diseases in man can be classified according to the anatomical site of primary involvement.
(a) Superficial Mycoses: The fungal infection is limited to outermost layers of skin and its appendages. The immune response in such infections is rarely induced or it is very mild.
(b) Cutaneous Mycoses: In this type the infection extends deeper into epidermis and it also invades hair and nails. The disease process may evoke high inflammatory response in the host.
(c) Subcutaneous Mycoses: The infection is due to organisms of low pathogenicity usually following traumatic injury. It involves dermis, subcutaneous tissues and sometimes muscles and fasciae. The causal agents of subcutaneous mycoses require mechanical introduction of organisms into tissues through traumatic implantation.
(d) Systemic Mycoses: The infection primarily involve a site like lungs and later on disseminates systemically to distant body sites. Systemic mycoses along with the opportunistic fungal infections, is called deep mycoses.
(e) Opportunistic Mycoses: Besides the above-mentioned four categories of fungal infections, fifth group has come into prominent focus because of increasing use of immunosuppressive therapy and pandemic of AIDS and is called opportunistic group. The infectious agents normally are of 12very low pathogenic potential, which produce full-fledged disease only under changed circumstances, mostly involving host debilitation.
The fungal infections are not transmitted sexually as commonly seen in viral, bacterial and even protozoal diseases. However, balanoposthitis caused by Candida species is supposed to be transmitted by the sexual contact. Moreover, piedra is also taken into this category as higher rates are reported among homosexuals in Denmark. In some of the fungal infections like histoplasmosis and mucormycosis, genital lesions have definitely been reported but their mode of infections is not considered to be through sexual route.
 
Significance of Fungi
The fungi have recently gained more importance due to increased incidence of various fungal diseases. Normally, most of these organisms are found as soil saprotrophic but whenever there is some underlying cause, the very same organism becomes significantly pathogenic. In addition to pathogenic potential, significance of fungi lies in the economy of nature is described below:
 
(a) Useful Properties of Fungi
The fungi are pathogenic to man, animals and plants causing various types of diseases. On the other hand it is also known that most of the fungi are beneficial to mankind. Their ability to break down complex organic substrate of almost every type thus contributing to recycling of carbon and other elements making fungi as an important component of our terrestrial ecosystem. The industrial uses of fungi as food, fermenter in food industry, producers of antibiotics, drugs, etc. are innumerable and may surpass damages, which these agents do as pathogens of man and animals. However, to date penicillin, lovastatin, cyclosporin, griseofulvin, cephalosporin and ergometrine are the most important pharmaceutical products that have been extracted from different fungal species.
The importance of fungi has been more emphasized towards beneficial effects, which are given below:
  • The edible fungi are widely used in industry as source of food, particularly wild or domesticated mushrooms of all shapes, sizes and colors, which belong to basidiomycetes. They are also put in salads, pizza and burgers.
  • The fungi are used to alter texture, improve flavor and increase palatability and digestibility of natural and processed foods. The yeast, Saccharomyces cerevisiae, is used in leavening of bread and other baked products. Hence it is known as Baker's yeast. The blue mold, Penicillium species, is used in ripening process to prepare speciality cheeses such as blue cheese.
  • Some of yeasts, Candida fukuyamaensis, are used to prepare special type of tea, known as Russian or Manchurian tea, which is presumed to be having some therapeutic potential in certain medical ailments. Hence it is consumed by many people under this impression. Candida fukuyamaensis is believed to be asexual state of Pichia guilliermondii.
  • At industrial level fungi are used to produce alcohol (Saccharomyces species), fat (Endomyces species) and proteins (Torulopsis species) through fermentation. Saccharomyces cerevisiae, also called Brewer's yeast, is used in the fermentation processes that result in production of beers, wines and spirits. All citric acid used in soft drinks, candies, artificial lemon juice, baked goods, etc. are produced industrially by fermentation by certain fungi like Aspergillus niger.
  • The fungi are now therapeutically being used as probiotics along with bacterial species like Lactobacillus and Bifidobacterium. In peculiar circumstances when the bacterial probiotic agents do not withstand onslaught of antimicrobials in antibiotic-associated diarrhoea, yeasts such as Saccharomyces boulardii can bear this assault thereby prove to be very effective in ameliorating such common alimentary ailments, while acting as probiotics. Hence S.boulardii is shown to be useful in the treatment (biotherapeutic agent) of acute infectious diarrhoea, the prevention or treatment of diarrhoea associated with antibiotic use and as an adjunctive therapy for Helicobacter pylori infection.
  • The discovery of popular antibiotic penicillin from Penicillium notatum (now called P.chrysogenum) by Alexander Fleming in 1928 was real breakthrough in history of medical sciences. In 1941, it revolutionized the basic approach in treating infectious diseases. P.chrysogenum is mold that is widely distributed in nature and is often found living on foods and indoor environments. Many fungi like P. chrysogenum are used as source of several β-lactam antibiotics, most significantly penicillin. Another fungus, Acremonium chrysogenum (also called Cephalosporium acremonium) is used in manufacturing cephalosporin. Cephalosporium chrysogenum was isolated for the first time by Thirumalachar and Sukapure from Pimpri, Pune, India.
  • The antifungal agent produced from fungi (Penicillium griseofulvin) is griseofulvin, is narrow-spectrum fungistatic antibiotic used for treating dermatophytes.
  • Aspergillus nidulans 13is used in making anidulafungin.
  • Fumagillin is produced from Aspergillus fumigatus and used in the treatment of microsporidiosis.
  • In addition to β-lactam antibiotics (penicillins and cephalosporins), fungi are used to produce immunosuppressant like cyclosporin A, which is a primary metabolite of several fungi, including Trichoderma polysporum and Cylindrocarpon lucidum.
  • The statin family of cholesterol-lowering drugs are derived from fungi. Simvastatin is prepared synthetically from fermentation product of Aspergillus terreus.
  • The ergot produced by Claviceps purpurea, a fungal pathogen of rye, is used for inducing uterine contractions, controlling bleeding and alleviating localized vascular disorders e.g. migraine. The ergot alkaloids belong to two groups: (a) amine alkaloid - ergometrine and (b) aminoacid alkaloids - ergotamine. Ergometrine or its semisynthetic analogue methylergometrine is clinically used for oxytocic effects. As ergotamine has α-adrenergic blocking and vasoconstriction properties, it is clinically used in treating migraine.
  • The fungi are now playing a vital role for producing natural products, most productive source of lead compounds in far reaching endeavor of new drug discovery. Epicoc-cum is known for its potential to produce diverse classes of biologically active secondary metabolites. Most of these fungal metabolites have cytotoxic, anticancer, antimicrobial and anti-diabetic activities.
  • A few fungal species (Saccharomyces cerevisiae, Neurospora crassa and Ustilago maydis) are studied as model organisms. Such studies are useful to acquire more knowledge of the basic processes involved in biochemistry, physiology, genetics and molecular biology and these results are applicable to many organisms. The dung fungus, Sordaria fimicola has recently been exploited in genetics studies.
  • The yeasts like Saccharomyces cerevisiae, Hansenula polymorpha and Pichia species have been used as heterogeneous host for preparation of recombinant vaccines for hepatitis B virus infection.
  • Fungi like Culicinomyces clavosporus have been used as traps and pathogen to control mosquitoes in malaria eradication. The entomopathogenic genus, Coelomomyces is also known to be pathogenic to larva and develop within larval hemocoel of mosquitoes. The Coelomomyces species are obligate parasitic fungi that alternate their saprotrophic and gametophytic stages between mosquitoes and copepods, respectively.
  • The pesticides of fungal origin have also been found to decrease malaria transmission in animal studies. After feeding on an infectious blood meal, mosquitoes have been exposed to surfaces coated with fungal biopesticides or entomopathogens, which have dramatically reduced their ability to transmit malaria.
  • Some of biotechnological applications of fungi especially in the field of food, agriculture, etc. are very significant. The biological measures have been applied in agriculture in Havana where some of fungi are used e.g. Beauveria bassiana, to control banana root borer, sweet potato weevil, rice water weevil and sugarcane borer. Verticillium lecanii specifically is bred to control sweet potato white fly, one of the most damaging pests in Cuba. Trichoderma species is used to control soil-borne diseases that attack tobacco, tomatoes and pepper.
  • Recently, one of the fungi (Gliocladium roseum), found prevalent in Ulmo tree in rain forests of Patagonia (South America), is tried to make biofuel. This digests the cellulose turning it into biofuel i.e. myco-diesel.
As an overall point of view, fungi have been used to cause man more satisfaction rather than trouble. They raised his bread, his wine and beer, carbonate his champagne, flavored his cheeses, furnished him edible mushrooms, produced drugs to stop bleeding, to reduce cholesterol, worked diligently to form antibiotics to help him fight bacterial as well as fungal infections and helped industry to produce many organics acids and other compounds. However, to a general mycologist they are even beautiful to behold and their unending variation is very fascinating, whereas to a medical mycologist they are now the dangerous and life-threatening organisms.
 
(b) Harmful Properties of Fungi
In addition to the useful properties, fungi are also important agents producing harmful effects, which are given below:
  • The pathogenic fungi are responsible for superficial, subcutaneous, systemic and opportunistic infections in man and animals.
  • Mycotoxicosis and mycetismus are becoming serious public health issues in most of the developing tropical and sub-tropical countries.
  • The fungi can spoil improperly stored agricultural produce like grain, foodstuff, vegetables and fruits. These are commonly seen in eatables as ‘bread molds’. The fungal pathogens are responsible for damages amounting about 10% of world's crops. It is estimated that as agents of plant disease, fungi cause annual crop losses of billions of dollars.
  • 14The fungi, especially molds, can cause decay of fabrics, timber, leather, electrical insulation and other synthetic materials. There may be extensive loss following failure to protect material from ravages of fungi in warm humid climates. It is also speculated that mutated fungi, which were breeding in space station Mir were responsible for the deterioration of its equipment.
  • The fungal growth on plastic products, especially on plasticize components, is known since 1940s. Each plastic product must be examined for its suitability to colonizing fungi. Saprotrophic fungi, especially in humid tropical areas, can colonize diskettes, audiotapes, videotapes and computer disks. The fungi belonging to genera like Alternaria, Aspergillus, Epicoccum, Paecilomyces, Penicillium and Trichoderma are observed on diskettes in the tropical countries.
 
Aeromycology
As fungal agents are part and parcel of soil and the environment, especially atmospheric air, which acts as the most common source of opportunistic as well as true fungal infections. The fungi in atmospheric air may be regularly monitored to know the magnitude of problem under which the population is living in an area. The fungal species constitute major component of air-spores. Various studies have revealed that fungal spores in air mostly come from fungi on plants or large fungal fruiting bodies that rise above soil surface and not from the ground. The soil itself is sinking into which most airborne spores finally disappear rather than their being a source. Many organisms found outside atmosphere will also be found inside buildings where they are introduced by air currents but main sources of contamination arise from within buildings from animals, humans or plants.
The aeromycology is study of airborne fungi, which is usually done with following purposes: (a) to know variety of fungi prevalent in particular region and also (b) to know seasonal variations of allergenic fungal spores. There are two methods for studying airborne fungi that may be used individually or concurrently; (i) slide exposure method and (ii) petri-dish exposure method. The combination of both these methods is ideal to minimize drawback of each other.
The value of aerobiology lies in the study of fungi that cause diseases to man and animals. Generally three types of diseases based on fungal origin are recognized: allergies, mycotoxicosis and mycoses. Hence inhalation of fungal spores and perhaps their metabolites may cause respiratory system disease such as bronchopulmonary aspergillosis, pulmonary mycotoxicosis and hypersensitivity reactions.
 
Surveillance of Fungal Infections
A regular monitoring of fungal infections is also required as it is done in case of viral, bacterial and parasitic infections. This is more important when large number of infections are healthcare-associated in nature and found prevalent in hospital environment. Moreover, these are found to be resistant to the most commonly used antifungal agents.
Outside hospital setup, surveillance of fungal infections is mandatory as certain outbreaks are reported in particular geographical areas. Some of diseases are prevalent in certain geographical pockets but in addition to that diseases are found prevalent in certain communities as well, which may be detected in time with watch of surveillance system.
Now molecular techniques are playing an important role not only in fungal diagnosis but in taxonomy and epidemiological aspects as well. These techniques have resolved many long-standing controversies on these aspects. Therefore, in any hospital set-up, regular surveillance of fungal infections is the most important, especially in epidemiological aspects of the healthcare-associated ones.
 
Safety in Medical Mycology Laboratory
There are certain fungal pathogens, which cause systemic infections associated with work related exposure to the staff working in diagnostic as well as research mycology laboratory. Four of these genera are established pathogenic fungi in which greatest potential of exposure is involved in such laboratories. These are: Coccidioides immitis, C.posadasii, Histoplasma capsulatum, Sporothrix schenckii and Cryptococcus neoformans. Additional species are being documented as etiological agents of mycotic morbidity and may pose potential threat for such type of infections. These agents usually take entry through respiratory route and may establish infection due to inhalation.
There are general safety measures like never smell or examine fungal culture in the open petri dish. The handling of clinical material as well as the fungal isolates should be done under biosafety hood. It is essential for both medical as well as classical mycologists to adhere 15standard safety measures during their laboratory work. The basic priority is to recognize potential risk of infection by these fungi. Secondly, acknowledgments that investigators have primary responsibility for containment of any organism with which personnel are working in laboratory to avoid any occupational health hazards. In diagnostic laboratory every clinical sample should be taken as highly infectious unless it is proven otherwise. Biosafety issues are not only important from medical point of view but because of legal angle also. Therefore, essential safety precautions and techniques should be adhered to in all research and diagnostic laboratories handling infective microorganisms including fungi. The general guidelines on laboratory safety practices are briefly described in Chapter 36.
The specifically recommended safety procedures and equipment are set out according to four Biohazard Safety Levels or BioSafety Levels (BSL) of laboratory facilities, dealing with potentially hazardous pathogens, moving through basic laboratories handling routine sample of low-risk microorganisms (BSL 1) to maximum containment laboratories working with highly infectious pathogens (BSL 4). These are also called corresponding levels of Containment (CL1 to 4), Category (Cat1 to 4) or Protection (P1 to 4) and the counterpart microorganisms as Hazardous Group (HG1 to 4).
A classification of fungi into biosafety categories is proposed and various criteria for attribution to Biosafety Levels have been laid down. The list of fungi stressing on ecological criteria is derived from these agents rather than host factors and include all species accepted in recent medical literature. Each fungal species is assigned a defined biohazard categories i.e. Biosafety Levels.
There are three primary goals for biological safety in medical mycology laboratory, which are as follows: (a) to reduce potential level of exposure of laboratory personnel; (b) to protect diagnostic and research work from exogenous contamination and (c) to prevent inadvertent contamination of environment outside laboratory.
The intent of dealing with this issue is to cover some of important aspects of biosafety; (i) standards in handling dimorphic fungal pathogens; (ii) principles and criteria of biosafety levels and classification of known medically important as well as environmental fungi according to their biosafety levels; (iii) medically important fungal waste and its safe disposal and (iv) biosafety and regulatory considerations in handling and mailing medically important fungi to a Reference Laboratory or Culture Collection.
The following safety procedures are adopted while working with potential fungal pathogens. These should be mandatory for all personnel posted in clinical or diagnostic laboratory. There are two most common means of getting infections by fungi in laboratory i.e. inhalation of conidia that are aerosolized and accidental inoculation through sharp object e.g. needles, scalpel blades and broken glasses while handling specimens.
The Biosafety Levels include laboratory practices and techniques, safety equipment and appropriate laboratory facilities and have been defined to guide those working with infectious agents. Keeping all types of microorganism in view, Centers for Disease Control and Prevention and National Institutes of Health, USA have divided Biosafety Levels into following categories, which are described below:
(a) Generally Regarded As Safe (GRAS): The harmless industrial strains like domesticated mutants widely applied in food production. GRAS refers to individual strains or clones whereas BSL refers to species in taxonomic sense.
(b) Biosafety Level 1: This level requires basic laboratory facilities and use of standard microbiological practices. It applies to use of specific microorganisms not known to cause disease in healthy human adults. These saprotrophic agents or plant pathogens are occupying non-vertebrate ecological niches or these are fungi utilizing dead animal products. The infections in this category are coincidental, superficial and non-invasive or mild in nature. Saccharomyces cerevisiae, which was categorized under GRAS, is now considered under this category because of substantial number of case reports by this organism.
(c) Biosafety Level 2: This level requires basic BSL 1 practices plus aprons, decontamination of all infectious wastes, limited access to laboratory, protective gloves and pasting of biohazard warning signs. This level is used when handling microorganisms of moderate-risk, which are associated with human diseases. These species are principally occupying non-vertebrate ecological niche but with relatively low ability to survive in vertebrate tissue. In severely immunocompromised patients they may cause deep or opportunistic mycoses. The pathogens causing superficial infections also fall into this category. Therefore, majority of fungi infecting man and animals are categorized under BSL 2.
Moreover, BSL 2 practices and facilities are recommended for handling and processing clinical specimens and identifying isolates of Coccidioides species. BSL 2
16practices differ from BSL 1 practices in that (i) laboratory personnel have specific training in handling pathogenic agents and are directed by qualified scientists; (ii) access to laboratory is limited when work is being conducted; (iii) extreme precautions are taken with contaminated sharp items and (iv) procedures in which infectious aerosols or splashes may be created, are conducted in Class II biological safety cabinets.
(d) Biosafety Level 3: This level requires BSL 2 facilities and practices supplemented by controlled access to laboratory and use of special laboratory clothing and containment equipment e.g. biological safety cabinet. Biosafety level 3 facilities require a separate negative pressure room. This level is used when potentiality of infection from aerosols, autoinoculation or ingestion exists and when concerned personnel are handling microorganisms which may cause severe or lethal infections. These pathogens are potentially able to cause severe or deep mycoses in otherwise healthy patients. Earlier list of fungi included in this category was mycelial forms of dimorphic fungi but now it also includes additional other potentially pathogenic fungi like Cladophialophora bantiana.
(e) Biosafety Level 4: This is maximum containment level consisting of work with dangerous and exotic agents, which have high potential for causing life-threatening diseases. It does not include any of fungal pathogens as most of the agents covered under this level are mainly viral in origin.
All Mycology Laboratories are covered under purview of two levels i.e. BSL 2 and at the most BSL 3. The clinical specimens that may contain fungi pathogenic to man should be handled by using BSL 2 practices, containment equipment and other facilities. There is need for clinical laboratories to adhere to safety standards promulgated by CDC Office of Health and Safety in 5th edition of ‘Biosafety in Microbiological and Biomedical Laboratories’ published in 2007. This document summarizes four recommended biosafety levels for infectious agents and addresses which types of biological safety cabinets are appropriate for each biosafety level and which are available online (See Appendix F).
The standard precautions include use of Biological Safety Cabinet or hood when working with clinical material suspected of having fungi. These cabinets are most commonly used primary containment devices in laboratories working with infectious agents. The staff must be properly trained in use of these cabinets. Therefore, biological safety cabinets are essential protective element for investigators working with infectious or potentially infectious fungal pathogens. The Class II biological safety cabinets have vertical laminar airflow with high efficiency particulate air (HEPA) filtered supply and exhaust air, which are 99.99% efficient in removing particles of size 0.3 μm or larger. An additional advantage of using biological safety cabinets is that they also protect clinical specimens from extraneous and airborne contamination. Hence they protect worker, product as well as the environment. The types of biosafety cabinets and their proper use have been described in details in 3rd edition of CDC-NIH publication entitled ‘Primary Containment for Biohazard: Selection, Installation and Use of Biological Safety Cabinets’ issued in September 2007.
BSL 2 procedures are specifically recommended for personnel working with clinical specimens, which may contain Blastomyces dermatitidis, Coccidioides immitis, C.posadasii, Histoplasma capsulatum, Cryptococcus neoformans, Sporothrix schenckii and pathogenic members of genera like Trichophyton, Microsporum and Epidermophyton. If culture of dimorphic fungus like Blastomyces, Coccidioides or Histoplasma is grown in its mycelial form, BSL 3 containment procedures should be adopted since conidia of these fungi have greater potential to cause infection through inhalation. In other words yeast (tissue) forms of some of dimorphic fungi are covered under BSL 2 and their counterpart mycelial forms strictly under provision of BSL 3 due to their potentially higher infectious nature.
 
Risk Groups
The biological agents are also categorized in Risk Groups (RG) based on their relative risk. Therefore, depending on the country or organization, this classification system may take the following factors into consideration: (i) Pathogenicity of the organism; (ii) Mode of transmission and host range; (iii) Availability of effective preventive measures like vaccines; (iv) Availability of effective treatment like antimicrobials. It is important to understand that biological agents are classified in a graded manner such that the level of hazard associated with RG1 being the lowest and RG4 being the highest. EHS Biosafety follows the NIH Guidelines categorization of Risk Groups as follows:
RG1: Are not associated with disease in healthy adult humans or animals.
RG2: Are associated with disease, which is rarely serious and for which preventative or therapeutic is often available.
RG3: Are associated with serious or lethal human disease for which preventative or therapeutics may be available.
RG4: 17Are associated with lethal human disease for which preventative or therapeutics are not readily available.
 
Outbreaks of Fungal Infections
Outbreaks of fungal infections following natural disasters and accidental/hospital acquisition are increasingly recognised. They have important public health and infection control consequences. Infections after natural disasters typically result from inhalation or cutaneous inoculation of environmental fungi. Clusters result from shared exposures. There have been >13 major global outbreaks in the past decade from air-borne, near-drowning or near-burial events encompassing fungi from Coccidioides immitis (n=3) to mucormycetes. Hurricane Katrina in 2008, the Boxing Day tsunami in 2004 and Joplin Tornado in 2011 ‘stand out’ with horrendous soft tissue injuries and disseminated infection with a mortality of 30-80%. Healthcare infections after disasters can follow e.g. Aspergillus meningitis from contaminated syringes (Sri Lankan tsunami). Case clusters also occur through use of contaminated pharmaceutical products, fomites and environmental hospital exposure. Mucormycosis has resulted from contaminated bed linen and tongue depressors. Fungal endophthalmitis (Fusarium, Bipolaris) followed minor eye surgery after irrigation with contaminated triamcinolone and Brilliant Blue Green. Moreover, there were 752 cases (64 deaths) of Exserohilum CNS and joint infections from contaminated methylprednisolone vials used to treat back pain in the USA with a national crisis. Improving physician and public health awareness of mycoses is essential with an urgent need for infrastructure for case reporting and a network of alerts.
 
Index of Suspicion
In routine medical practice, the infectious diseases are usually considered to be caused by bacteria, viruses and seldom by the parasites. Fungus, as such the cause of infectious disease is rarely considered. Therefore, to tackle such infections, often antibacterial antibiotic are prescribed and underlying pathogenesis of fungal infection goes on increasing. By the time realization is there about the fungal infection, it is too late and situation becomes almost irreversible. Therefore, fungal infections should be considered as the differential diagnosis, wherever, a slightest doubt of fungal involvement is there. If the index of suspicion is kept very high then the additional diagnostic methodology is adopted along with the routine protocol and final diagnosis is reached without wastage of valuable time thereby proper antifungal treatment is instituted and life of the patient is saved. Hence a high index of suspicion is the key to an early diagnosis of fungal diseases, which is must to tackle such life-threatening infections.
 
Medical Mycology in India
In India, fungal infections are known since ancient civilization and have been mentioned in Aryan documents such as Atharva Veda, wherein mycetoma is described as Padavalmikam i.e. anthill foot. This finding was observed by John Gill in 1842 in Madurai district of a south Indian state, Tamil Nadu, which was subsequently designated as ‘madura foot’ by Henry Carter in 1860.
In the beginning of 20th century, School of Tropical Medicine and Hygiene was established during the British regimen in 1920s in Kolkata (then Calcutta), the eastern zone of country. There was separate Department of Medical Mycology to cater services in this field of medical sciences, where Prof. Anisetti Thammayya did his innovative work in this field. In the western zone, Prof. K R Joshi consolidated different aspects of medical mycology, especially in the field of mycetoma and opportunistic fungal infections. Similarly, in southern zone, Prof. A S Thambiah, Prof. A Kamalam, Prof. Pankajalakshmi V Venugopal and Prof. P A Thomas in Tamil Nadu and Prof. B M Hemashettar in Karnataka, have brought Medical Mycology in the country to the level of substantial recognition. In the central India, mycological services were consolidated by Prof. S M Singh and his colleagues.
In northern side, Department of Medical Mycology was established at Vallabhbhai Patel Chest Institute, Delhi in 1959 to provide research, diagnostic, therapeutic mycological services in the national capital and its surrounding areas. Prof. H S Randhawa and Prof. H C Gugnani further developed this Department and currently being looked after by Prof. Anuradha Chowdhary. During the same period Mycology Sections were developed in All India Institute of Medical Sciences (AIIMS) and National Institute of Communicable Diseases (NICD), both in New Delhi. Prof. L N Mohapatra did pioneering work in this field at AIIMS, which was continued by Prof. Uma Banerjee and her colleagues.
The Division of Mycology at Postgraduate Institute of Medical Sciences and Research, Chandigarh was started under enthusiastic leadership of Prof. Pushpa Talwar in 1964 and is at present being looked after by Prof. Arunaloke Chakrabarti. This is one of the leading centers of Medical Mycology and providing diagnostic services as well as 18conducting research activities. Some of Indian institutions, where active work is currently going on, have been mentioned in Appendix E of this Textbook.
A curriculum was designed by Prof. R. Sambasiva and colleagues in 1999, wherein it is emphasized that postgraduates in Microbiology must have basic knowledge of theory and practical aspects in Medical Mycology, along with its other branches. In India, this branch of Microbiology has now attained substantially sound footing. The following salient features are also relevant to describe on this issue:
  • Previously there were only a few case reports from India about fungal infections but now many prospective studies are being conducted mainly based on epidemiology and diagnostic modalities in the field of Medical Mycology.
  • The international leading organizations in this field like ISHAM are helping medical institutions in India especially for training of staff, who handle difficulties related to management of fungal infections.
  • The first edition of Textbook of Medical Mycology was published on July 1, 1995 which has modified trends in understanding of Medical Mycology at the grass root level in India and its neighboring countries followed by second edition in April 2002 and third edition in October 2008. It is expected that this fourth edition (2017) will also be able to further consolidate its endeavor in the times to come.
  • The national body of Indian scientists interested in field of medical mycology came into existence. The Society of Indian Human and Animal Mycologists (SIHAM) was founded in February 1996 in its First Conference held at Jabalpur, Madhya Pradesh. Actually idea of making national body of mycologists was conceived in 1994 at time of 12th ISHAM Congress held at Adelaide (Australia), which was materialized after two years under the dynamic leadership of Prof. S M Singh, in the form of SIHAM, which is actively working for enhancing the cause of Medical Mycology in India. The National Conferences of SIHAM are being regularly held after a gap of two years. The very 1st Conference was held in Jabalpur (1996), 2nd in Jodhpur (1998), 3rd in Chennai (2000), 4th in Annamalai Nagar (2002), 5th in Chandigarh (2004), 6th in Hyderabad (2006), 7th in Mumbai (2008), 8th in New Delhi (2010), 9th in Siliguri (2012), 10th in Coimbatore (2014) and 11th in Shimla (2016). Now, 12th National SIHAM Conference will be held under the leadership of Dr. Jayanthi Savio in February 2018 at Bangalore (Karnataka).
  • Many Seminars, Symposia, Workshops, Continuing Medical Education (CME) and Professional Development Programs (PDP) are being held in this field from time to time in different parts of the country. The relevant documents of these events are referred in Appendix F as Bibliography. A number of monographs and proceedings of these meetings are now available dealing with various aspects of Medical Mycology to provide firm footing in India.
  • During this course of development, Mycology Reference Laboratories (MRL) are supposed to be designated to provide basic facilities for identification of fungi, teaching of graduates and postgraduates, other academic and service-oriented infrastructure in this unique field of medical sciences. The Indian Council of Medical Research (ICMR) has recognized the Division of Mycology as the ‘Centre for Advanced Research in Medical Mycology’ in the year 2005. It is providing two ICMR-WHO-PGI Training Courses in Diagnostic Medical Mycology in a calendar year: (i) summer course for the young faculty and medical scientists (May-June) and (ii) winter course for technical staff (December). In addition, it is the national reference centre for the identification, national culture collection as well as antifungal susceptibility testing of medically significant pathogenic fungi.
  • The quality control in diagnostic mycology is essential to maintain standard in imparting the services. This may be internal or external quality control. In India periodic exercise was started by VPCI, New Delhi in the form of Proficiency Testing as external quality control. This service is now being provided as EQAS by the PGIMER, Chandigarh and dealt briefly in Appendix E. Therefore, auditing of Medical Mycology services is mandatory to impart good quality laboratory practices.
 
Fungal Repositories
The Budapest Treaty on international recognition of deposit of microorganisms for purposes of patent procedure was done at Budapest on April 28, 1977 and was amended on September 26, 1980. In India, based on these guidelines, the Microbial Type Culture Collection Center and Gene Bank (MTCC), IMTECH, Chandigarh was established and is functioning since October 4, 2002. In addition to other microorganisms, it is repository for the fungi, mainly isolated from the environment. The National Culture Collection of Pathogenic Fungi (NCCPF) was set-up at the PGIMER, 19Chandigarh by the ICMR in 2010, where the fungal isolates can be deposited. The details of such issues are dealt in Appendix E.
 
Fungal Registries
In the recent times, lot of websites have been created which register data pertaining to any of the fungal issues, analyze it and periodically publish it from time to time. According to the feedback received, corrective measures are also taken. Some of the registries are: Mucormycetes (www.zygomyco.net), Emerging Fungal Infections (www.fungiscope.net), Trichosporon (www.trireg.com) and research collaboration focused on pediatric invasive fungal infections i.e. International Pediatric Fungal Network (www.ipfn.org), etc.
Further Reading
  1. Abdel-Azeem AM.The history, fungal biodiversity, conservation and future perspectives for mycology in Egypt. IMA Fungus. 2010; 1: 123-42.
  1. Adams RI, Miletto M, Taylor JW, et al. The diversity and distribution of fungi on residential surfaces. PLoS One. 2013; 8: e78866.PMID: 24223861.
  1. Akritidis N.Parasitic, fungal and prion zoonoses: An expanding universe of candidates for human disease. Clin Microbiol Infect. 2011; 17: 331-5.
  1. Alvarez Duarte E, Denning DW.Serious fungal infections in Chile. Eur J Clin Microbiol Infect Dis. 2017; 36: 983-6.
  1. Antinori S, Nebuloni M, Magni C, et al. Trends in the postmortem diagnosis of opportunistic invasive fungal infections in patients with AIDS: A retrospective study of 1,630 autopsies performed between 1984 and 2002. Am J Clin Pathol. 2009; 132: 221-7.
  1. Antinori S, Peri AM, Milazzo L.Fungal meningitis in England and Wales. Lancet Infect Dis. 2014; 14: 921.
  1. Aridogan IA, Izol V, Ilkit M.Superficial fungal infections of the male genitalia: A review. Crit Rev Microbiol. 2011; 37: 237-44.
  1. Armstrong-James D, Bicanic T, Brown GD, et al.AIDS-related mycoses: Current progress in the field and future priorities. Trends Microbiol. 2017; 25: 428-30.
  1. Armstrong-James D, Meintjes G, Brown GD.A neglected epidemic: Fungal infections in HIV/AIDS. Trends Microbiol. 2014; 22: 120-7.
  1. Arnold C.Fungal meningitis outbreak affects over 7000. Lancet Neurol. 2013; 12: 429-30.
  1. Badiane AS, Ndiaye D, Denning DW.Burden of fungal infections in Senegal. Mycoses. 2015; 58 (Suppl. 5):63-9.
  1. Bassetti M, Righi E.Overview of fungal infections - The Italian experience. Semin Respir Crit Care Med. 2015; 36: 796-805.
  1. Batac MC, Denning D.Serious fungal infections in the Philippines. Eur J Clin Microbiol Infect Dis. 2017; 36: 937-41.
  1. Beardsley J, Denning DW, Chau NV, et al.Estimating the burden of fungal disease in Vietnam. Mycoses. 2015; 58 (Suppl. 5):101-6.
  1. Ben R, Denning DW.Estimating the burden of fungal diseases in Israel. Isr Med Assoc J. 2015; 17: 374-9.
  1. Bidartondo MI, Read DJ, Trappe JM, et al.The dawn of symbiosis between plants and fungi. Biol Lett. 2011; 7: 574-7.
  1. Binder U, Lass-Florl C.Epidemiology of invasive fungal infections in the Mediterranean area. Mediterr J Hematol Infect Dis. 2011; 3: e20110016.
  1. Blackwell M.The fungi: 1, 2, 3 …… 5.1 million species? Am J Bot. 2011; 98: 426-38.
  1. Brandt ME, Park BJ.Think fungus—Prevention and control of fungal infections. Emerg Infect Dis. 2013; 19: 1688-9.
  1. Brown GD, Denning DW, Levitz SM.Tackling human fungal infections. Science. 2012; 336: 647.
  1. Brown GD, Denning DW, Gow NA, et al.Hidden killers: Human fungal infections. Sci Transl Med. 2012; 4:165rv13.
  1. Bustamante B, Denning DW, Campos PE.Serious fungal infections in Peru. Eur J Clin Microbiol Infect Dis. 2017; 36: 943-8.
  1. Casadevall A, Pirofski LA. Exserohilum rostratum fungal meningitis associated with methylprednisolone injections. Future Microbiol. 2013; 8: 135-7.
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